Atrial septal defect sinus venosus
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-In-Chief: Priyamvada Singh, M.B.B.S. [2]; Cafer Zorkun, M.D., Ph.D. [3] Assistant Editor(s)-In-Chief: Kristin Feeney, B.S. [4]
Overview
During fetal development, the septal wall may fail to fuse causing an atrial septal defect to arise. A sinus venosus atrial septal defect is one such type of malformation arising from the irregular development of the septum and the superior vena cava or the inferior vena cava.
Sinus Venosus Atrial Septal Defect
- In this type of atrial septal defect the common wall between the coronary sinus and the left atrium is not developed.
- Some patients with coronary sinus atrial septal defects may also have a persistent left superior vena cava.
- A sinus venosus ASD is a type of atrial septum defect in which the defect in the septum involves the venous inflow of either the superior vena cava or the inferior vena cava.
- A sinus venosus ASD that involves the superior vena cava makes up 2 to 3% of all interatrial communication. It is located at the junction of the superior vena cava and the right atrium. It is frequently associated with anomalous drainage of the right-sided pulmonary veins into the right atrium (instead of the normal drainage of the pulmonary veins into the left atrium).[1]
Echocardiography in Sinus Venosus Defects
Medical Therapy
Indications for Surgical Repair
Surgical Closure
- sinus venosus ASD is commonly found to be associated with partial anomalous pulmonary venous return.
- In presence of partial anomalous pulmonary venous return (pulmonary veins draining into the right atrium), atrial septal defect is corrected by placement of patch over the defect. Proper care should be taken so that the anomalous pulmonary venous drainage is diverted into the left atrium.
- Improper pulmonary venous return due to redirection can cause pulmonary venous hypertension.
References
- ↑ Davia J, Cheitlin M, Bedynek J (1973). "Sinus venosus atrial septal defect: analysis of fifty cases". Am Heart J. 85 (2): 177–85. PMID 4569755.