Cirrhosis differential diagnosis
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] ; Associate Editor(s)-in-Chief: Aditya Govindavarjhulla, M.B.B.S. [2]
Overview
Differentiating Cirrhosis from other Diseases
| Condition | Differentiating Signs and Symptoms | Differentiating Tests |
| Constrictive pericarditis | Increased jugular venous pressure, atrial fibrillation, and tachycardia. Quiet heart sounds with a third heart sound (ventricular knock) present. | *EKG will show tachycardia, atrial fibrillation, low-voltage QRS complexes and T wave abnormalities. Doppler ultrasound will show ventricular filling abnormalities. |
| Budd-Chiari Syndrome | Delayed onset reaction caused by IgG or IgM antibodies, directed at drug-hapten coated cells. | Thrombocytopenia, anemia, cytopenia. |
| Splenic vein thrombosis | Delayed in onset and caused by IgG immune complex formation and deposition, and complement activation. | Vasculitis, serum sickness, arthralgia, fever, rash. |
| Portal vein thrombosis | The presentation of drug molecules by major histocompatability complexes to T cells, causing the release of cytokines and other inflammatory mediators. Also associated with the activation of eosinophils, monocytes, and neutrophils. | rashes, organ tissue damage, contact sensitivity. |
| Schistosomiasis | The presentation of drug molecules by major histocompatability complexes to T cells, causing the release of cytokines and other inflammatory mediators. Also associated with the activation of eosinophils, monocytes, and neutrophils. | rashes, organ tissue damage, contact sensitivity. |
| Sarcoidosis | The presentation of drug molecules by major histocompatability complexes to T cells, causing the release of cytokines and other inflammatory mediators. Also associated with the activation of eosinophils, monocytes, and neutrophils. | rashes, organ tissue damage, contact sensitivity. |
| Inferior vena cava obstruction | The presentation of drug molecules by major histocompatability complexes to T cells, causing the release of cytokines and other inflammatory mediators. Also associated with the activation of eosinophils, monocytes, and neutrophils. | rashes, organ tissue damage, contact sensitivity. |