Nephrogenic diabetes insipidus pathophysiology
Nephrogenic diabetes insipidus Microchapters |
Differentiating Nephrogenic diabetes insipidus from other Diseases |
---|
Diagnosis |
Treatment |
Case Studies |
Nephrogenic diabetes insipidus pathophysiology On the Web |
American Roentgen Ray Society Images of Nephrogenic diabetes insipidus pathophysiology |
Directions to Hospitals Treating Nephrogenic diabetes insipidus |
Risk calculators and risk factors for Nephrogenic diabetes insipidus pathophysiology |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor in Chief: Cafer Zorkun, M.D., Ph.D. [2]
Please help WikiDoc by adding more content here. It's easy! Click here to learn about editing.
Overview
Pathophysiology
Nephrogenic diabetes insipidus (NDI) is characterized by inability to concentrate the urine, which results in polyuria (excessive urine production) and polydipsia (excessive thirst). Affected untreated infants usually have poor feeding and failure to thrive, and rapid onset of severe dehydration with illness, hot environment, or the withholding of water. Short stature and secondary dilatation of the ureters and bladder from the high urine volume is common in untreated individuals.