Strychnine poisoning history and symptoms

Revision as of 19:32, 31 January 2013 by Kalsang Dolma (talk | contribs) (Created page with "__NOTOC__ {{Strychnine poisoning}} Please help WikiDoc by adding content here. It's easy! Click here to learn about editing. {{CMG}}''' Associ...")
(diff) ← Older revision | Latest revision (diff) | Newer revision → (diff)
Jump to navigation Jump to search

Strychnine Poisoning Microchapters

Home

Patient Information

Overview

Pathophysiology

Causes

Differentiating Strychnine Poisoning from other Diseases

Epidemiology and Demographics

Risk Factors

Natural History, Complications and Prognosis

Diagnosis

History and Symptoms

Physical Examination

Laboratory Findings

Other Imaging Findings

Other Diagnostic Studies

Treatment

Medical Therapy

Primary Prevention

Secondary Prevention

Cost-Effectiveness of Therapy

Future or Investigational Therapies

Case Studies

Case #1

Strychnine poisoning history and symptoms On the Web

Most recent articles

Most cited articles

Review articles

CME Programs

Powerpoint slides

Images

American Roentgen Ray Society Images of Strychnine poisoning history and symptoms

All Images
X-rays
Echo & Ultrasound
CT Images
MRI

Ongoing Trials at Clinical Trials.gov

US National Guidelines Clearinghouse

NICE Guidance

FDA on Strychnine poisoning history and symptoms

CDC on Strychnine poisoning history and symptoms

Strychnine poisoning history and symptoms in the news

Blogs on Strychnine poisoning history and symptoms

Directions to Hospitals Treating Strychnine poisoning

Risk calculators and risk factors for Strychnine poisoning history and symptoms

Please help WikiDoc by adding content here. It's easy! Click here to learn about editing.

Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] Associate Editor-In-Chief: Cafer Zorkun, M.D., Ph.D. [2]

Overview

Ten to twenty minutes after exposure, the body's muscles begin to spasm, starting with the head and neck. The spasms then spread to every muscle in the body, with nearly continuous convulsions, and get worse at the slightest stimulus. The convulsions progress, increasing in intensity and frequency until the backbone arches continually. Death comes from asphyxiation caused by paralysis of the neural pathways that control breathing, or by exhaustion from the convulsions. The subject will die within 2–3 hours after exposure. At the point of death, the body "freezes" immediately, even in the middle of a convulsion, resulting in instantaneous rigor mortis.

References


Template:WikiDoc Sources