Ogilvie syndrome laboratory findings
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Ahmed Elsaiey, MBBCH [2]
Overview
There are no specific diagnostic laboratory findings associated with Ogilvie's syndrome. The laboratory findings may include leukocytosis due to the underlying disease not due to the pseudo-obstruction itself. Many patients with Ogilvie syndrome may have metabolic imbalance which include hypokalemiaand hypocalcemia. Other laboratory tests that can be performed to exclude other causes include complete blood count, lactate levels, and thyroid hormonelevels.
Laboratory Findings
- There are no specific diagnostic laboratory findings associated with Ogilvie's syndrome. The laboratory findings may include leukocytosis due to the underlying disease not due to the pseudo-obstruction itself.[1]
- In most of the patients metabolic imbalance may occur and it includes:[2][3]
- General laboratory tests are performed to exclude other causes and disease. The laboratory tests include the following:
- To exclude acute abdominal pain diagnosis in patients suspected with perforation, the following laboratory tests are performed:
- In patients with diarrhea, the following laboratory tests are performed:[4]
References
- ↑ Vanek VW, Al-Salti M (1986). "Acute pseudo-obstruction of the colon (Ogilvie's syndrome). An analysis of 400 cases". Dis Colon Rectum. 29 (3): 203–10. PMID 3753674.
- ↑ Jetmore AB, Timmcke AE, Gathright JB, Hicks TC, Ray JE, Baker JW (1992). "Ogilvie's syndrome: colonoscopic decompression and analysis of predisposing factors". Dis Colon Rectum. 35 (12): 1135–42. PMID 1473414.
- ↑ Sandle GI, Hunter M (2010). "Apical potassium (BK) channels and enhanced potassium secretion in human colon". QJM. 103 (2): 85–9. doi:10.1093/qjmed/hcp159. PMID 19892809.
- ↑ Simon M, Duong JP, Mallet V, Jian R, MacLennan KA, Sandle GI; et al. (2008). "Over-expression of colonic K+ channels associated with severe potassium secretory diarrhoea after haemorrhagic shock". Nephrol Dial Transplant. 23 (10): 3350–2. doi:10.1093/ndt/gfn411. PMID 18653901.