Rapidly progressive glomerulonephritis risk factors: Difference between revisions
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{{Rapidly progressive glomerulonephritis}} | {{Rapidly progressive glomerulonephritis}} | ||
{{CMG}}; {{AE}} {{JSS}} | |||
==Overview== | |||
Common risk factors in the development of rapidly progressive glomerulonephritis may be occupational, environmental, [[genetic]], and [[viral]]. | |||
== | ==Risk Factors== | ||
Common risk factors in the development of rapidly progressive glomerulonephritis may be occupational, environmental, [[genetic]], and [[viral]]. <ref name="pmid24456936">{{cite journal| author=Hellmark T, Segelmark M| title=Diagnosis and classification of Goodpasture's disease (anti-GBM). | journal=J Autoimmun | year= 2014 | volume= 48-49 | issue= | pages= 108-12 | pmid=24456936 | doi=10.1016/j.jaut.2014.01.024 | pmc= | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=24456936 }} </ref><ref name="pmid1536151">{{cite journal| author=Bombassei GJ, Kaplan AA| title=The association between hydrocarbon exposure and anti-glomerular basement membrane antibody-mediated disease (Goodpasture's syndrome). | journal=Am J Ind Med | year= 1992 | volume= 21 | issue= 2 | pages= 141-53 | pmid=1536151 | doi= | pmc= | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=1536151 }} </ref> | |||
=== Common risk factrors === | |||
* Pre existing [[renal disease]]. | |||
* Presence of [[HLA]]DRB1, HLA DP1, DQ and DRB4s<ref name="pmid15652778">{{cite journal| author=Jagiello P, Gross WL, Epplen JT| title=Complex genetics of Wegener granulomatosis. | journal=Autoimmun Rev | year= 2005 | volume= 4 | issue= 1 | pages= 42-7 | pmid=15652778 | doi=10.1016/j.autrev.2004.06.003 | pmc= | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=15652778 }} </ref> | |||
* [[Upper respiratory tract infection]] | |||
* [[Sepsis]] | |||
* Tobacco smoking | |||
* Intravenous drug abuse | |||
=== Less common risk factors === | |||
* Cocaine use | |||
* Exposure to hydrocarbons( formaldehyde) | |||
* HIgh concenteration of FiO<sub>2</sub> | |||
==References== | |||
{{reflist|2}} | |||
{{WH}} | {{WH}} | ||
{{WS}} | {{WS}} | ||
[[Category:Autoimmune diseases]] | |||
[[Category:Kidney diseases]] | |||
[[Category:Rheumatology]] | |||
[[Category:Pulmonology]] | |||
[[Category:Needs content]] | |||
[[Category:Disease]] |
Latest revision as of 19:32, 24 July 2018
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Jogeet Singh Sekhon, M.D. [2]
Overview
Common risk factors in the development of rapidly progressive glomerulonephritis may be occupational, environmental, genetic, and viral.
Risk Factors
Common risk factors in the development of rapidly progressive glomerulonephritis may be occupational, environmental, genetic, and viral. [1][2]
Common risk factrors
- Pre existing renal disease.
- Upper respiratory tract infection
- Sepsis
- Tobacco smoking
- Intravenous drug abuse
Less common risk factors
- Cocaine use
- Exposure to hydrocarbons( formaldehyde)
- HIgh concenteration of FiO2
References
- ↑ Hellmark T, Segelmark M (2014). "Diagnosis and classification of Goodpasture's disease (anti-GBM)". J Autoimmun. 48-49: 108–12. doi:10.1016/j.jaut.2014.01.024. PMID 24456936.
- ↑ Bombassei GJ, Kaplan AA (1992). "The association between hydrocarbon exposure and anti-glomerular basement membrane antibody-mediated disease (Goodpasture's syndrome)". Am J Ind Med. 21 (2): 141–53. PMID 1536151.
- ↑ Jagiello P, Gross WL, Epplen JT (2005). "Complex genetics of Wegener granulomatosis". Autoimmun Rev. 4 (1): 42–7. doi:10.1016/j.autrev.2004.06.003. PMID 15652778.