Beriberi differential diagnosis: Difference between revisions
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{{Beriberi}} | {{Beriberi}} | ||
{{CMG}}; {{AE}}{{AIA}} | {{CMG}}; {{AE}}{{AIA}} | ||
==Overview== | ==Overview== | ||
Beriberi should be differentiated from other causes of cardiac dysfunction (wet beriberi), peripheral neuropathy (dry beriberi), delirium ( | Beriberi should be differentiated from other causes of cardiac dysfunction (wet beriberi), peripheral neuropathy (dry beriberi), delirium (Wernicke-Korsakoff syndrome) andexcessive alcohol drinking. However, the differential diagnosis is broad due to the non-specific symptoms of cardiac and neural involvement associated with the condition. | ||
==Differentiating Beriberi from other Diseases== | ==Differentiating Beriberi from other Diseases== | ||
Beriberi should be differentiated from other causes of cardiac dysfunction (wet beriberi), peripheral neuropathy (dry beriberi), delirium (WKS), and other disorders caused by excessive alcohol drinking. However, the differential diagnosis is broad due to the non-specific symptoms of cardiac and neural involvement associated with beriberi.<ref name="pmid30151974">{{cite journal| author=Whitfield KC, Bourassa MW, Adamolekun B, Bergeron G, Bettendorff L, Brown KH et al.| title=Thiamine deficiency disorders: diagnosis, prevalence, and a roadmap for global control programs. | journal=Ann N Y Acad Sci | year= 2018 | volume= 1430 | issue= 1 | pages= 3-43 | pmid=30151974 | doi=10.1111/nyas.13919 | pmc=6392124 | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=30151974 }}</ref> | Beriberi should be differentiated from other causes of cardiac dysfunction (wet beriberi), peripheral neuropathy (dry beriberi), delirium (WKS), and other disorders caused by excessive alcohol drinking. However, the differential diagnosis is broad due to the non-specific symptoms of cardiac and neural involvement associated with beriberi.<ref name="pmid30151974">{{cite journal| author=Whitfield KC, Bourassa MW, Adamolekun B, Bergeron G, Bettendorff L, Brown KH et al.| title=Thiamine deficiency disorders: diagnosis, prevalence, and a roadmap for global control programs. | journal=Ann N Y Acad Sci | year= 2018 | volume= 1430 | issue= 1 | pages= 3-43 | pmid=30151974 | doi=10.1111/nyas.13919 | pmc=6392124 | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=30151974 }}</ref><ref name="pmid29360523">{{cite journal| author=DiNicolantonio JJ, Liu J, O'Keefe JH| title=Thiamine and Cardiovascular Disease: A Literature Review. | journal=Prog Cardiovasc Dis | year= 2018 | volume= 61 | issue= 1 | pages= 27-32 | pmid=29360523 | doi=10.1016/j.pcad.2018.01.009 | pmc= | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=29360523 }}</ref><ref name="pmid30281514">{{cite journal| author=Chandrakumar A, Bhardwaj A, 't Jong GW| title=Review of thiamine deficiency disorders: Wernicke encephalopathy and Korsakoff psychosis. | journal=J Basic Clin Physiol Pharmacol | year= 2018 | volume= 30 | issue= 2 | pages= 153-162 | pmid=30281514 | doi=10.1515/jbcpp-2018-0075 | pmc= | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=30281514 }}</ref> | ||
{| class="wikitable" | {| class="wikitable" | ||
|+ | |+ | ||
!Disorders | ! align="center" style="background:#4479BA; color: #FFFFFF;" + |Disorders | ||
!Etiology | ! align="center" style="background:#4479BA; color: #FFFFFF;" + |Etiology | ||
!Clinical Presentation | ! align="center" style="background:#4479BA; color: #FFFFFF;" + |Clinical Presentation | ||
!Laboratory findings | ! align="center" style="background:#4479BA; color: #FFFFFF;" + |Laboratory findings | ||
|- | |- | ||
!Cardiomyopathy due to other causes as alcohol or DM | |||
| | | | ||
* Idiopathic | * Idiopathic | ||
| Line 32: | Line 31: | ||
* Elevated [[troponin]] (in ischemia) | * Elevated [[troponin]] (in ischemia) | ||
|- | |- | ||
!Delirium and delusional disorders | |||
| | | | ||
* [[Acute liver failure]] | * [[Acute liver failure]] | ||
| Line 49: | Line 48: | ||
* Glucose and electrolyte levels | * Glucose and electrolyte levels | ||
|- | |- | ||
!Nerve entrapment disorders | |||
|Chronic injuries to nerves as they pass between bones and ligaments: | |Chronic injuries to nerves as they pass between bones and ligaments: | ||
| Line 68: | Line 67: | ||
* MRI short inversion imaging recovery (STIR) technique | * MRI short inversion imaging recovery (STIR) technique | ||
|- | |- | ||
!Alcoholic hepatitis | |||
| | | | ||
* Chronic and excessive alcohol consumption | * Chronic and excessive alcohol consumption | ||
| Line 82: | Line 81: | ||
* Liver US: Changes in liver size and dilatation of hepatic veins. | * Liver US: Changes in liver size and dilatation of hepatic veins. | ||
|- | |- | ||
!Diabetic ketoacidosis | |||
|In patients with type 1 DM, exposed to: | |In patients with type 1 DM, exposed to: | ||
| Line 102: | Line 101: | ||
* Impaired renal function | * Impaired renal function | ||
|- | |- | ||
!Hyperthyroidism | |||
| | | | ||
* Primary: [[Graves' disease|Grave's disease]], toxic thyroid nodules and adenoma | * Primary: [[Graves' disease|Grave's disease]], toxic thyroid nodules and adenoma | ||
| Line 121: | Line 120: | ||
* Thyroid stimulating antibodies: Elevated only in Grave's disease | * Thyroid stimulating antibodies: Elevated only in Grave's disease | ||
|- | |- | ||
!Folic acid deficiency | |||
| | | | ||
*[[Malnutrition]] | *[[Malnutrition]] | ||
Latest revision as of 20:02, 24 January 2020
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Beriberi Microchapters |
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Beriberi differential diagnosis On the Web |
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Risk calculators and risk factors for Beriberi differential diagnosis |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Abdelrahman Ibrahim Abushouk, MD[2]
Overview
Beriberi should be differentiated from other causes of cardiac dysfunction (wet beriberi), peripheral neuropathy (dry beriberi), delirium (Wernicke-Korsakoff syndrome) andexcessive alcohol drinking. However, the differential diagnosis is broad due to the non-specific symptoms of cardiac and neural involvement associated with the condition.
Differentiating Beriberi from other Diseases
Beriberi should be differentiated from other causes of cardiac dysfunction (wet beriberi), peripheral neuropathy (dry beriberi), delirium (WKS), and other disorders caused by excessive alcohol drinking. However, the differential diagnosis is broad due to the non-specific symptoms of cardiac and neural involvement associated with beriberi.[1][2][3]
| Disorders | Etiology | Clinical Presentation | Laboratory findings |
|---|---|---|---|
| Cardiomyopathy due to other causes as alcohol or DM |
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| Delirium and delusional disorders |
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| Nerve entrapment disorders | Chronic injuries to nerves as they pass between bones and ligaments:
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Within the distribution of the affected nerve, the patient may complain of:
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| Alcoholic hepatitis |
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| Diabetic ketoacidosis | In patients with type 1 DM, exposed to:
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| Hyperthyroidism |
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| Folic acid deficiency |
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References
- ↑ Whitfield KC, Bourassa MW, Adamolekun B, Bergeron G, Bettendorff L, Brown KH; et al. (2018). "Thiamine deficiency disorders: diagnosis, prevalence, and a roadmap for global control programs". Ann N Y Acad Sci. 1430 (1): 3–43. doi:10.1111/nyas.13919. PMC 6392124. PMID 30151974.
- ↑ DiNicolantonio JJ, Liu J, O'Keefe JH (2018). "Thiamine and Cardiovascular Disease: A Literature Review". Prog Cardiovasc Dis. 61 (1): 27–32. doi:10.1016/j.pcad.2018.01.009. PMID 29360523.
- ↑ Chandrakumar A, Bhardwaj A, 't Jong GW (2018). "Review of thiamine deficiency disorders: Wernicke encephalopathy and Korsakoff psychosis". J Basic Clin Physiol Pharmacol. 30 (2): 153–162. doi:10.1515/jbcpp-2018-0075. PMID 30281514.