Silicosis classification: Difference between revisions
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__NOTOC__ | __NOTOC__ | ||
{{Silicosis}} | {{Silicosis}} | ||
{{CMG}} | {{CMG}} {{AE}} {{AV}} | ||
==Overview== | ==Overview== | ||
There are 5 subtypes of silicosis that are differentiated according to the duration of exposure, development of symptoms, pulmonary function, and findings on chest imaging. Silicosis is classified as either simple chronic, complicated chronic, interstitial pulmonary fibrosis, accelerated silicosis, | There are 5 subtypes of silicosis that are differentiated according to the duration of exposure, development of symptoms, pulmonary function, and findings on chest imaging. Silicosis is classified as either simple chronic, complicated chronic, interstitial pulmonary fibrosis, accelerated silicosis, or acute silicosis. | ||
==Classification== | ==Classification== | ||
There are 5 subtypes of silicosis that are differentiated according to the duration of exposure, development of symptoms, pulmonary function, and findings on chest imaging. | There are 5 subtypes of silicosis that are differentiated according to the duration of exposure, development of symptoms, pulmonary function, and findings on chest imaging.<ref name="pmid25479706">{{cite journal| author=Fernández Álvarez R, Martínez González C, Quero Martínez A, Blanco Pérez JJ, Carazo Fernández L, Prieto Fernández A| title=Guidelines for the diagnosis and monitoring of silicosis. | journal=Arch Bronconeumol | year= 2015 | volume= 51 | issue= 2 | pages= 86-93 | pmid=25479706 | doi=10.1016/j.arbres.2014.07.010 | pmc= | url=http://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=25479706 }} </ref> | ||
===Simple Chronic=== | |||
*Respiratory disease often develops > 10 years following exposure to low - moderate concentrations of silica dust. | |||
*Patients usually remain asymptomatic, making the diagnosis of silicosis very difficult. | |||
*Pulmonary function tests are typically normal but may also be reduced. | |||
*On chest imaging, [[nodules]] are < 10 mm in diameter. | |||
===Complicated Chronic=== | |||
*Respiratory disease often develops > 10 years following exposure to silica dust. | |||
*Patients may report [[dyspnea]] and [[cough]]. | |||
*[[Pulmonary function tests|Pulmonary function]] may be either normal or reduced. | |||
*On chest imaging, nodules > 1 cm with either obstructive or restrictive changes of variable severity | |||
===Interstitial Pulmonary Fibrosis=== | |||
*Respiratory disease often develops > 10 years following exposure to silica dust. | |||
*Patients may report [[dyspnea]] and [[cough]]. | |||
*Pulmonary function may be either normal or reduced. | |||
*On chest imaging, diffuse reticulonodular pattern similar to [[idiopathic pulmonary fibrosis]] is usually observed. | |||
===Accelerated Silicosis=== | |||
*Respiratory disease often develops 5 to 10 years following exposure to silica dust. | |||
*Patients often report [[dyspnea]], [[weakness]], and [[weight loss]]. | |||
*Pulmonary function is often reduced with rapid deterioration of [[FVC]] and [[FEV1]]. | |||
*On chest imaging, rapidly progressive nodules and masses are observed. | |||
===Acute Silicosis=== | |||
*Respiratory disease often develops within 5 years of exposure to extremely high concentrations of silica dust. | |||
*Patients often report severe, disabling [[dyspnea]], [[weakness]], [[weight loss]], and progressive respiratory failure. | |||
*Pulmonary function is usually reduced with restrictive changes and reduced diffusion capacity. | |||
*On chest imaging, bilateral perihilar acinar-pattern [[consolidations]] with [[ground glass appearance]] is observed. | |||
==References== | ==References== | ||
{{Reflist|2}} | {{Reflist|2}} | ||
[[Category:Pulmonology]] | [[Category:Pulmonology]] | ||
{{WH}} | {{WH}} | ||
{{WS}} | {{WS}} | ||
Latest revision as of 15:25, 8 June 2016
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] Associate Editor(s)-in-Chief: Aparna Vuppala, M.B.B.S. [2]
Overview
There are 5 subtypes of silicosis that are differentiated according to the duration of exposure, development of symptoms, pulmonary function, and findings on chest imaging. Silicosis is classified as either simple chronic, complicated chronic, interstitial pulmonary fibrosis, accelerated silicosis, or acute silicosis.
Classification
There are 5 subtypes of silicosis that are differentiated according to the duration of exposure, development of symptoms, pulmonary function, and findings on chest imaging.[1]
Simple Chronic
- Respiratory disease often develops > 10 years following exposure to low - moderate concentrations of silica dust.
- Patients usually remain asymptomatic, making the diagnosis of silicosis very difficult.
- Pulmonary function tests are typically normal but may also be reduced.
- On chest imaging, nodules are < 10 mm in diameter.
Complicated Chronic
- Respiratory disease often develops > 10 years following exposure to silica dust.
- Patients may report dyspnea and cough.
- Pulmonary function may be either normal or reduced.
- On chest imaging, nodules > 1 cm with either obstructive or restrictive changes of variable severity
Interstitial Pulmonary Fibrosis
- Respiratory disease often develops > 10 years following exposure to silica dust.
- Patients may report dyspnea and cough.
- Pulmonary function may be either normal or reduced.
- On chest imaging, diffuse reticulonodular pattern similar to idiopathic pulmonary fibrosis is usually observed.
Accelerated Silicosis
- Respiratory disease often develops 5 to 10 years following exposure to silica dust.
- Patients often report dyspnea, weakness, and weight loss.
- Pulmonary function is often reduced with rapid deterioration of FVC and FEV1.
- On chest imaging, rapidly progressive nodules and masses are observed.
Acute Silicosis
- Respiratory disease often develops within 5 years of exposure to extremely high concentrations of silica dust.
- Patients often report severe, disabling dyspnea, weakness, weight loss, and progressive respiratory failure.
- Pulmonary function is usually reduced with restrictive changes and reduced diffusion capacity.
- On chest imaging, bilateral perihilar acinar-pattern consolidations with ground glass appearance is observed.
References
- ↑ Fernández Álvarez R, Martínez González C, Quero Martínez A, Blanco Pérez JJ, Carazo Fernández L, Prieto Fernández A (2015). "Guidelines for the diagnosis and monitoring of silicosis". Arch Bronconeumol. 51 (2): 86–93. doi:10.1016/j.arbres.2014.07.010. PMID 25479706.