Silicosis classification: Difference between revisions

Jump to navigation Jump to search
No edit summary
mNo edit summary
 
(5 intermediate revisions by 2 users not shown)
Line 1: Line 1:
__NOTOC__
__NOTOC__
{{Silicosis}}
{{Silicosis}}
{{CMG}} {{AE}}
{{CMG}} {{AE}} {{AV}}


==Overview==
==Overview==
Line 7: Line 7:


==Classification==
==Classification==
There are 5 subtypes of silicosis that are differentiated according to the duration of exposure, development of symptoms, pulmonary function, and findings on chest imaging.<ref name="pmid17400659">{{cite journal| author=Arakawa H, Johkoh T, Honma K, Saito Y, Fukushima Y, Shida H et al.| title=Chronic interstitial pneumonia in silicosis and mix-dust pneumoconiosis: its prevalence and comparison of CT findings with idiopathic pulmonary fibrosis. | journal=Chest | year= 2007 | volume= 131 | issue= 6 | pages= 1870-6 | pmid=17400659 | doi=10.1378/chest.06-2553 | pmc= | url=http://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=17400659 ; }} </ref><ref name="pmid11836467">{{cite journal| author=Park R, Rice F, Stayner L, Smith R, Gilbert S, Checkoway H| title=Exposure to crystalline silica, silicosis, and lung disease other than cancer in diatomaceous earth industry workers: a quantitative risk assessment. | journal=Occup Environ Med | year= 2002 | volume= 59 | issue= 1 | pages= 36-43 | pmid=11836467 | doi= | pmc=PMC1740205 | url=http://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=11836467 ; }} </ref><ref name="pmid17976433">{{cite journal| author=Greenberg MI, Waksman J, Curtis J| title=Silicosis: a review. | journal=Dis Mon | year= 2007 | volume= 53 | issue= 8 | pages= 394-416 | pmid=17976433 | doi=10.1016/j.disamonth.2007.09.020 | pmc= | url=http://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=17976433 ; }} </ref>
There are 5 subtypes of silicosis that are differentiated according to the duration of exposure, development of symptoms, pulmonary function, and findings on chest imaging.<ref name="pmid25479706">{{cite journal| author=Fernández Álvarez R, Martínez González C, Quero Martínez A, Blanco Pérez JJ, Carazo Fernández L, Prieto Fernández A| title=Guidelines for the diagnosis and monitoring of silicosis. | journal=Arch Bronconeumol | year= 2015 | volume= 51 | issue= 2 | pages= 86-93 | pmid=25479706 | doi=10.1016/j.arbres.2014.07.010 | pmc= | url=http://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=25479706  }} </ref>
===Simple Chronic===
===Simple Chronic===
*Respiratory disease often develops > 10 years following exposure to low - moderate concentrations of silica dust.
*Respiratory disease often develops > 10 years following exposure to low - moderate concentrations of silica dust.
*Patients usually remain asymptomatic, making the diagnosis of silicosis very difficult.
*Patients usually remain asymptomatic, making the diagnosis of silicosis very difficult.
*Pulmonary function tests are typically normal but may also be reduced.
*Pulmonary function tests are typically normal but may also be reduced.
*On chest imaging, nodules are < 10 mm in diameter.
*On chest imaging, [[nodules]] are < 10 mm in diameter.


===Complicated Chronic===
===Complicated Chronic===
*Respiratory disease often develops > 10 years following exposure to silica dust.
*Respiratory disease often develops > 10 years following exposure to silica dust.
*Patients may report dyspnea and cough.
*Patients may report [[dyspnea]] and [[cough]].
*Pulmonary function may be either normal or reduced.
*[[Pulmonary function tests|Pulmonary function]] may be either normal or reduced.
*On chest imaging, nodules > 1 cm with either obstructive or restrictive changes of variable severity
*On chest imaging, nodules > 1 cm with either obstructive or restrictive changes of variable severity


===Interstitial Pulmonary Fibrosis===
===Interstitial Pulmonary Fibrosis===
*Respiratory disease often develops > 10 years following exposure to silica dust.
*Respiratory disease often develops > 10 years following exposure to silica dust.
**Patients may report dyspnea and cough.
*Patients may report [[dyspnea]] and [[cough]].
*Pulmonary function may be either normal or reduced.
*Pulmonary function may be either normal or reduced.
*On chest imaging, diffuse reticulonodular pattern similar to idiopathic pulmonary fibrosis is usually observed.
*On chest imaging, diffuse reticulonodular pattern similar to [[idiopathic pulmonary fibrosis]] is usually observed.


===Accelerated Silicosis===
===Accelerated Silicosis===
*Respiratory disease often develops 5 to 10 years following exposure to silica dust.
*Respiratory disease often develops 5 to 10 years following exposure to silica dust.
*Patients often report dyspnea, weakness, and weight loss.
*Patients often report [[dyspnea]], [[weakness]], and [[weight loss]].
*Pulmonary function is often reduced with rapid deterioration of FVC and FEV1.
*Pulmonary function is often reduced with rapid deterioration of [[FVC]] and [[FEV1]].
*On chest imaging, rapidly progressive nodules and masses are observed.
*On chest imaging, rapidly progressive nodules and masses are observed.


===Acute Silicosis===
===Acute Silicosis===
*Respiratory disease often develops within 5 years of exposure to extremely high concentrations of silica dust.
*Respiratory disease often develops within 5 years of exposure to extremely high concentrations of silica dust.
*Patients often report severe, disabling dyspnea, weakness, weight loss, and progressive respiratory failure.
*Patients often report severe, disabling [[dyspnea]], [[weakness]], [[weight loss]], and progressive respiratory failure.
*Pulmonary function is usually reduced with restrictive changes and reduced diffusion capacity.
*Pulmonary function is usually reduced with restrictive changes and reduced diffusion capacity.
*On chest imaging, bilateral perihilar acinar-pattern consolidations with ground glass appearance is observed.
*On chest imaging, bilateral perihilar acinar-pattern [[consolidations]] with [[ground glass appearance]] is observed.


==References==
==References==
{{Reflist|2}}


{{Reflist|2}}
[[Category:Pulmonology]]
[[Category:Pulmonology]]
[[Category:Occupational diseases]]


{{WH}}
{{WH}}
{{WS}}
{{WS}}

Latest revision as of 15:25, 8 June 2016

Silicosis Microchapters

Home

Patient Information

Overview

Historical Perspective

Classification

Pathophysiology

Causes

Differentiating Silicosis from other Diseases

Epidemiology and Demographics

Risk Factors

Screening

Natural History, Complications and Prognosis

Diagnosis

Diagnostic Criteria

History and Symptoms

Physical Examination

Laboratory Findings

Chest X Ray

CT

MRI

Other Imaging Findings

Other Diagnostic Studies

Treatment

Medical Therapy

Surgery

Primary Prevention

Secondary Prevention

Cost-Effectiveness of Therapy

Future or Investigational Therapies

Case Studies

Case #1

Silicosis classification On the Web

Most recent articles

Most cited articles

Review articles

CME Programs

Powerpoint slides

Images

American Roentgen Ray Society Images of Silicosis classification

All Images
X-rays
Echo & Ultrasound
CT Images
MRI

Ongoing Trials at Clinical Trials.gov

US National Guidelines Clearinghouse

NICE Guidance

FDA on Silicosis classification

CDC on Silicosis classification

Silicosis classification in the news

Blogs on Silicosis classification

Directions to Hospitals Treating Silicosis

Risk calculators and risk factors for Silicosis classification

Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] Associate Editor(s)-in-Chief: Aparna Vuppala, M.B.B.S. [2]

Overview

There are 5 subtypes of silicosis that are differentiated according to the duration of exposure, development of symptoms, pulmonary function, and findings on chest imaging. Silicosis is classified as either simple chronic, complicated chronic, interstitial pulmonary fibrosis, accelerated silicosis, or acute silicosis.

Classification

There are 5 subtypes of silicosis that are differentiated according to the duration of exposure, development of symptoms, pulmonary function, and findings on chest imaging.[1]

Simple Chronic

  • Respiratory disease often develops > 10 years following exposure to low - moderate concentrations of silica dust.
  • Patients usually remain asymptomatic, making the diagnosis of silicosis very difficult.
  • Pulmonary function tests are typically normal but may also be reduced.
  • On chest imaging, nodules are < 10 mm in diameter.

Complicated Chronic

  • Respiratory disease often develops > 10 years following exposure to silica dust.
  • Patients may report dyspnea and cough.
  • Pulmonary function may be either normal or reduced.
  • On chest imaging, nodules > 1 cm with either obstructive or restrictive changes of variable severity

Interstitial Pulmonary Fibrosis

  • Respiratory disease often develops > 10 years following exposure to silica dust.
  • Patients may report dyspnea and cough.
  • Pulmonary function may be either normal or reduced.
  • On chest imaging, diffuse reticulonodular pattern similar to idiopathic pulmonary fibrosis is usually observed.

Accelerated Silicosis

  • Respiratory disease often develops 5 to 10 years following exposure to silica dust.
  • Patients often report dyspnea, weakness, and weight loss.
  • Pulmonary function is often reduced with rapid deterioration of FVC and FEV1.
  • On chest imaging, rapidly progressive nodules and masses are observed.

Acute Silicosis

  • Respiratory disease often develops within 5 years of exposure to extremely high concentrations of silica dust.
  • Patients often report severe, disabling dyspnea, weakness, weight loss, and progressive respiratory failure.
  • Pulmonary function is usually reduced with restrictive changes and reduced diffusion capacity.
  • On chest imaging, bilateral perihilar acinar-pattern consolidations with ground glass appearance is observed.

References

  1. Fernández Álvarez R, Martínez González C, Quero Martínez A, Blanco Pérez JJ, Carazo Fernández L, Prieto Fernández A (2015). "Guidelines for the diagnosis and monitoring of silicosis". Arch Bronconeumol. 51 (2): 86–93. doi:10.1016/j.arbres.2014.07.010. PMID 25479706.

Template:WH Template:WS