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{{CMG}} {{AE}} {{Sahar}} | {{CMG}} {{AE}} {{Sahar}} | ||
==Overview== | ==Overview== | ||
The most potent [[risk factor]] in the development of secondary amyloidosis is a persistent [[inflammatory]] [[disorders]]. | The most potent [[risk factor]] in the development of secondary amyloidosis is a persistent [[inflammatory]] [[disorders]]. Chronic [[infections]] and [[inflammatory]] [[arthritis]] are among the most common [[risk factors]]. | ||
==Risk Factors== | ==Risk Factors== | ||
* The most potent [[risk factor]] in the development of secondary amyloidosis is a persistent [[inflammatory]] [[disorders]].<ref name="KoivuniemiPaimela2009">{{cite journal|last1=Koivuniemi|first1=Riitta|last2=Paimela|first2=Leena|last3=Suomalainen|first3=Risto|last4=Törnroth|first4=Tom|last5=Leirisalo-Repo|first5=Marjatta|title=Amyloidosis is frequently undetected in patients with rheumatoid arthritis|journal=Amyloid|volume=15|issue=4|year=2009|pages=262–268|issn=1350-6129|doi=10.1080/13506120802524676}}</ref> | * The most potent [[risk factor]] in the development of secondary amyloidosis is a persistent [[inflammatory]] [[disorders]].<ref name="KoivuniemiPaimela2009">{{cite journal|last1=Koivuniemi|first1=Riitta|last2=Paimela|first2=Leena|last3=Suomalainen|first3=Risto|last4=Törnroth|first4=Tom|last5=Leirisalo-Repo|first5=Marjatta|title=Amyloidosis is frequently undetected in patients with rheumatoid arthritis|journal=Amyloid|volume=15|issue=4|year=2009|pages=262–268|issn=1350-6129|doi=10.1080/13506120802524676}}</ref> | ||
* | * Chronic [[infections]] and [[inflammatory]] [[arthritis]] are among the most common [[risk factors]].<ref name="BlankHegenbart2018">{{cite journal|last1=Blank|first1=Norbert|last2=Hegenbart|first2=Ute|last3=Dietrich|first3=Sascha|last4=Brune|first4=Maik|last5=Beimler|first5=Jörg|last6=Röcken|first6=Christoph|last7=Müller-Tidow|first7=Carsten|last8=Lorenz|first8=Hanns-Martin|last9=Schönland|first9=Stefan O.|title=Obesity is a significant susceptibility factor for idiopathic AA amyloidosis|journal=Amyloid|volume=25|issue=1|year=2018|pages=37–45|issn=1350-6129|doi=10.1080/13506129.2018.1429391}}</ref><ref name="van der HilstYamada2008">{{cite journal|last1=van der Hilst|first1=J. C. H.|last2=Yamada|first2=T.|last3=Op den Camp|first3=H. J. M.|last4=van der Meer|first4=J. W. M.|last5=Drenth|first5=J. P. H.|last6=Simon|first6=A.|title=Increased susceptibility of serum amyloid A 1.1 to degradation by MMP-1: potential explanation for higher risk of type AA amyloidosis|journal=Rheumatology|volume=47|issue=11|year=2008|pages=1651–1654|issn=1462-0324|doi=10.1093/rheumatology/ken371}}</ref><ref name="PapaDoglio2017">{{cite journal|last1=Papa|first1=Riccardo|last2=Doglio|first2=Matteo|last3=Lachmann|first3=Helen J.|last4=Ozen|first4=Seza|last5=Frenkel|first5=Joost|last6=Simon|first6=Anna|last7=Neven|first7=Bénédicte|last8=Kuemmerle-Deschner|first8=Jasmin|last9=Ozgodan|first9=Huri|last10=Caorsi|first10=Roberta|last11=Federici|first11=Silvia|last12=Finetti|first12=Martina|last13=Trachana|first13=Maria|last14=Brunner|first14=Jurgen|last15=Bezrodnik|first15=Liliana|last16=Pinedo Gago|first16=Mari Carmen|last17=Maggio|first17=Maria Cristina|last18=Tsitsami|first18=Elena|last19=Al Suwairi|first19=Wafaa|last20=Espada|first20=Graciela|last21=Shcherbina|first21=Anna|last22=Aksu|first22=Guzide|last23=Ruperto|first23=Nicolino|last24=Martini|first24=Alberto|last25=Ceccherini|first25=Isabella|last26=Gattorno|first26=Marco|title=A web-based collection of genotype-phenotype associations in hereditary recurrent fevers from the Eurofever registry|journal=Orphanet Journal of Rare Diseases|volume=12|issue=1|year=2017|issn=1750-1172|doi=10.1186/s13023-017-0720-3}}</ref> | ||
* Other possible [[risk factors]] include: | * Other possible [[risk factors]] include: | ||
** [[Obesity]] | **[[Obesity]] | ||
** Aging | ** Aging | ||
** SAA1 gene alleles | ** SAA1 [[gene]] alleles | ||
** Monogenic [[periodic fever syndromes]], such as: | ** Monogenic [[Periodic fever syndrome|periodic fever syndromes]], such as: | ||
*** [[FMF]] | ***[[FMF]] | ||
*** [[TNF Receptor associated periodic syndrome]] ([[TRAPS]]) | ***[[TNF receptor associated periodic syndrome|TNF Receptor associated periodic syndrome]] ([[TRAPS]]) | ||
* [[Conditions]] associated | ***[[CAPS|Cryopyrin-associated periodic fever syndrome]] | ||
***[[Mevalonate kinase deficiency]] | |||
**[[Tuberculosis]] | |||
**[[Leprosy]] | |||
** Whipple Disease | |||
**[[Osteomyelitis]] | |||
**[[Chronic pyelonephritis]] | |||
**[[Subacute bacterial endocarditis]] | |||
** Chronic cutaneous ulcers | |||
*[[Conditions]] Predisposing to chronic [[infections]] include: | |||
**[[Cystic fibrosis]] | |||
**[[Bronchiectasis]] | |||
**[[Kartagener syndrome]] | |||
**[[Epidermolysis bullosa]] | |||
** Injected drug abuse | |||
**[[Jejuno-ileal bypass]] | |||
**[[Paraplegia]] | |||
**[[Sickle cell anemia]] | |||
**[[Immunodeficiency]] | |||
**[[Common variable immunodeficiency]] | |||
**[[Cyclic neutropenia]] | |||
**[[Hyperimmunoglobulin M syndrome]] | |||
**[[Hypogammaglobulinemia]] | |||
** Sex-linked agammaglobulinemia | |||
**[[Human Immunodeficiency Virus|Human immunodeficiency virus]]/[[AIDS]] | |||
**[[Neoplasia]] | |||
**[[Adenocarcinoma]] | |||
**[[Basal cell carcinoma]] | |||
**[[Carcinoid tumors|Carcinoid tumor]] | |||
**[[Castleman disease]] | |||
**[[Gastrointestinal stromal tumor]] | |||
**[[Hairy cell leukemia]] | |||
**[[Hepatic adenoma]] | |||
**[[Hodgkin disease]] | |||
**[[Mesothelioma]] | |||
**[[Renal cell carcinoma]] | |||
**[[Sarcoma]] | |||
** Inflammatory Arthritis | |||
** Adult-onset Still disease | |||
**[[Ankylosing spondylitis]] | |||
**[[Juvenile idiopathic arthritis]] | |||
**[[Psoriatic arthropathy]] | |||
**[[Reiter's syndrome|Reiter syndrome]] | |||
**[[Rheumatoid arthritis]] | |||
**[[Gout]] | |||
** Systemic [[Vasculitis]] | |||
** Antineutrophil cytoplasmic antibody-associated vasculitis | |||
**[[Behcet disease]] | |||
**[[Giant cell arteritis]] | |||
**[[Polyarteritis nodosa]] | |||
**[[Polymyalgia rheumatica]] | |||
**[[Systemic lupus erythematosus]] | |||
**[[Takayasu arteritis]] | |||
**[[Inflammatory Bowel Disease]] | |||
**[[Ulcerative colitis]] | |||
**[[Crohn disease]] | |||
* Others include: | |||
**[[Atrial myxoma]] | |||
** Inflammatory abdominal aortic aneurism | |||
**[[Retroperitoneal fibrosis]] | |||
**[[SAPHO syndrome|SAPHO (synovitis, acne, pustulosis, hyperostosis, and osteitis) syndrome]] | |||
**[[Sarcoidosis]] | |||
**[[Sinus histiocytosis with massive lymphadenopathy]] | |||
==References== | |||
{{Reflist|2}} | |||
Latest revision as of 14:00, 30 October 2019
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] Associate Editor(s)-in-Chief: Sahar Memar Montazerin, M.D.[2]
Overview
The most potent risk factor in the development of secondary amyloidosis is a persistent inflammatory disorders. Chronic infections and inflammatory arthritis are among the most common risk factors.
Risk Factors
- The most potent risk factor in the development of secondary amyloidosis is a persistent inflammatory disorders.[1]
- Chronic infections and inflammatory arthritis are among the most common risk factors.[2][3][4]
- Other possible risk factors include:
- Obesity
- Aging
- SAA1 gene alleles
- Monogenic periodic fever syndromes, such as:
- Tuberculosis
- Leprosy
- Whipple Disease
- Osteomyelitis
- Chronic pyelonephritis
- Subacute bacterial endocarditis
- Chronic cutaneous ulcers
- Conditions Predisposing to chronic infections include:
- Cystic fibrosis
- Bronchiectasis
- Kartagener syndrome
- Epidermolysis bullosa
- Injected drug abuse
- Jejuno-ileal bypass
- Paraplegia
- Sickle cell anemia
- Immunodeficiency
- Common variable immunodeficiency
- Cyclic neutropenia
- Hyperimmunoglobulin M syndrome
- Hypogammaglobulinemia
- Sex-linked agammaglobulinemia
- Human immunodeficiency virus/AIDS
- Neoplasia
- Adenocarcinoma
- Basal cell carcinoma
- Carcinoid tumor
- Castleman disease
- Gastrointestinal stromal tumor
- Hairy cell leukemia
- Hepatic adenoma
- Hodgkin disease
- Mesothelioma
- Renal cell carcinoma
- Sarcoma
- Inflammatory Arthritis
- Adult-onset Still disease
- Ankylosing spondylitis
- Juvenile idiopathic arthritis
- Psoriatic arthropathy
- Reiter syndrome
- Rheumatoid arthritis
- Gout
- Systemic Vasculitis
- Antineutrophil cytoplasmic antibody-associated vasculitis
- Behcet disease
- Giant cell arteritis
- Polyarteritis nodosa
- Polymyalgia rheumatica
- Systemic lupus erythematosus
- Takayasu arteritis
- Inflammatory Bowel Disease
- Ulcerative colitis
- Crohn disease
- Others include:
- Atrial myxoma
- Inflammatory abdominal aortic aneurism
- Retroperitoneal fibrosis
- SAPHO (synovitis, acne, pustulosis, hyperostosis, and osteitis) syndrome
- Sarcoidosis
- Sinus histiocytosis with massive lymphadenopathy
References
- ↑ Koivuniemi, Riitta; Paimela, Leena; Suomalainen, Risto; Törnroth, Tom; Leirisalo-Repo, Marjatta (2009). "Amyloidosis is frequently undetected in patients with rheumatoid arthritis". Amyloid. 15 (4): 262–268. doi:10.1080/13506120802524676. ISSN 1350-6129.
- ↑ Blank, Norbert; Hegenbart, Ute; Dietrich, Sascha; Brune, Maik; Beimler, Jörg; Röcken, Christoph; Müller-Tidow, Carsten; Lorenz, Hanns-Martin; Schönland, Stefan O. (2018). "Obesity is a significant susceptibility factor for idiopathic AA amyloidosis". Amyloid. 25 (1): 37–45. doi:10.1080/13506129.2018.1429391. ISSN 1350-6129.
- ↑ van der Hilst, J. C. H.; Yamada, T.; Op den Camp, H. J. M.; van der Meer, J. W. M.; Drenth, J. P. H.; Simon, A. (2008). "Increased susceptibility of serum amyloid A 1.1 to degradation by MMP-1: potential explanation for higher risk of type AA amyloidosis". Rheumatology. 47 (11): 1651–1654. doi:10.1093/rheumatology/ken371. ISSN 1462-0324.
- ↑ Papa, Riccardo; Doglio, Matteo; Lachmann, Helen J.; Ozen, Seza; Frenkel, Joost; Simon, Anna; Neven, Bénédicte; Kuemmerle-Deschner, Jasmin; Ozgodan, Huri; Caorsi, Roberta; Federici, Silvia; Finetti, Martina; Trachana, Maria; Brunner, Jurgen; Bezrodnik, Liliana; Pinedo Gago, Mari Carmen; Maggio, Maria Cristina; Tsitsami, Elena; Al Suwairi, Wafaa; Espada, Graciela; Shcherbina, Anna; Aksu, Guzide; Ruperto, Nicolino; Martini, Alberto; Ceccherini, Isabella; Gattorno, Marco (2017). "A web-based collection of genotype-phenotype associations in hereditary recurrent fevers from the Eurofever registry". Orphanet Journal of Rare Diseases. 12 (1). doi:10.1186/s13023-017-0720-3. ISSN 1750-1172.