Secondary amyloidosis risk factors: Difference between revisions

Jump to navigation Jump to search
← Older edit
VisualWikitext
Sahar Memar Montazerin (talk | contribs)
Bureaucrats, bureaucrats, nocaptcha, smwadministrator, Administrators
10,191 edits
No edit summary
Sahar Memar Montazerin (talk | contribs)
Bureaucrats, bureaucrats, nocaptcha, smwadministrator, Administrators
10,191 edits
No edit summary
 
(4 intermediate revisions by the same user not shown)
Line 3: Line 3:
{{CMG}} {{AE}} {{Sahar}}
{{CMG}} {{AE}} {{Sahar}}
==Overview==
==Overview==
The most potent [[risk factor]] in the development of secondary amyloidosis is a persistent [[inflammatory]] [[disorders]].
The most potent [[risk factor]] in the development of secondary amyloidosis is a persistent [[inflammatory]] [[disorders]]. Chronic [[infections]] and [[inflammatory]] [[arthritis]] are among the most common [[risk factors]].
==Risk Factors==
==Risk Factors==
* The most potent [[risk factor]] in the development of secondary amyloidosis is a persistent [[inflammatory]] [[disorders]].<ref name="KoivuniemiPaimela2009">{{cite journal|last1=Koivuniemi|first1=Riitta|last2=Paimela|first2=Leena|last3=Suomalainen|first3=Risto|last4=Törnroth|first4=Tom|last5=Leirisalo-Repo|first5=Marjatta|title=Amyloidosis is frequently undetected in patients with rheumatoid arthritis|journal=Amyloid|volume=15|issue=4|year=2009|pages=262–268|issn=1350-6129|doi=10.1080/13506120802524676}}</ref>
* The most potent [[risk factor]] in the development of secondary amyloidosis is a persistent [[inflammatory]] [[disorders]].<ref name="KoivuniemiPaimela2009">{{cite journal|last1=Koivuniemi|first1=Riitta|last2=Paimela|first2=Leena|last3=Suomalainen|first3=Risto|last4=Törnroth|first4=Tom|last5=Leirisalo-Repo|first5=Marjatta|title=Amyloidosis is frequently undetected in patients with rheumatoid arthritis|journal=Amyloid|volume=15|issue=4|year=2009|pages=262–268|issn=1350-6129|doi=10.1080/13506120802524676}}</ref>
* chronic [[infections]] and [[inflammatory]] [[arthritis]] are among the most common [[risk factors]].<ref name="BlankHegenbart2018">{{cite journal|last1=Blank|first1=Norbert|last2=Hegenbart|first2=Ute|last3=Dietrich|first3=Sascha|last4=Brune|first4=Maik|last5=Beimler|first5=Jörg|last6=Röcken|first6=Christoph|last7=Müller-Tidow|first7=Carsten|last8=Lorenz|first8=Hanns-Martin|last9=Schönland|first9=Stefan O.|title=Obesity is a significant susceptibility factor for idiopathic AA amyloidosis|journal=Amyloid|volume=25|issue=1|year=2018|pages=37–45|issn=1350-6129|doi=10.1080/13506129.2018.1429391}}</ref><ref name="van der HilstYamada2008">{{cite journal|last1=van der Hilst|first1=J. C. H.|last2=Yamada|first2=T.|last3=Op den Camp|first3=H. J. M.|last4=van der Meer|first4=J. W. M.|last5=Drenth|first5=J. P. H.|last6=Simon|first6=A.|title=Increased susceptibility of serum amyloid A 1.1 to degradation by MMP-1: potential explanation for higher risk of type AA amyloidosis|journal=Rheumatology|volume=47|issue=11|year=2008|pages=1651–1654|issn=1462-0324|doi=10.1093/rheumatology/ken371}}</ref><ref name="PapaDoglio2017">{{cite journal|last1=Papa|first1=Riccardo|last2=Doglio|first2=Matteo|last3=Lachmann|first3=Helen J.|last4=Ozen|first4=Seza|last5=Frenkel|first5=Joost|last6=Simon|first6=Anna|last7=Neven|first7=Bénédicte|last8=Kuemmerle-Deschner|first8=Jasmin|last9=Ozgodan|first9=Huri|last10=Caorsi|first10=Roberta|last11=Federici|first11=Silvia|last12=Finetti|first12=Martina|last13=Trachana|first13=Maria|last14=Brunner|first14=Jurgen|last15=Bezrodnik|first15=Liliana|last16=Pinedo Gago|first16=Mari Carmen|last17=Maggio|first17=Maria Cristina|last18=Tsitsami|first18=Elena|last19=Al Suwairi|first19=Wafaa|last20=Espada|first20=Graciela|last21=Shcherbina|first21=Anna|last22=Aksu|first22=Guzide|last23=Ruperto|first23=Nicolino|last24=Martini|first24=Alberto|last25=Ceccherini|first25=Isabella|last26=Gattorno|first26=Marco|title=A web-based collection of genotype-phenotype associations in hereditary recurrent fevers from the Eurofever registry|journal=Orphanet Journal of Rare Diseases|volume=12|issue=1|year=2017|issn=1750-1172|doi=10.1186/s13023-017-0720-3}}</ref>
* Chronic [[infections]] and [[inflammatory]] [[arthritis]] are among the most common [[risk factors]].<ref name="BlankHegenbart2018">{{cite journal|last1=Blank|first1=Norbert|last2=Hegenbart|first2=Ute|last3=Dietrich|first3=Sascha|last4=Brune|first4=Maik|last5=Beimler|first5=Jörg|last6=Röcken|first6=Christoph|last7=Müller-Tidow|first7=Carsten|last8=Lorenz|first8=Hanns-Martin|last9=Schönland|first9=Stefan O.|title=Obesity is a significant susceptibility factor for idiopathic AA amyloidosis|journal=Amyloid|volume=25|issue=1|year=2018|pages=37–45|issn=1350-6129|doi=10.1080/13506129.2018.1429391}}</ref><ref name="van der HilstYamada2008">{{cite journal|last1=van der Hilst|first1=J. C. H.|last2=Yamada|first2=T.|last3=Op den Camp|first3=H. J. M.|last4=van der Meer|first4=J. W. M.|last5=Drenth|first5=J. P. H.|last6=Simon|first6=A.|title=Increased susceptibility of serum amyloid A 1.1 to degradation by MMP-1: potential explanation for higher risk of type AA amyloidosis|journal=Rheumatology|volume=47|issue=11|year=2008|pages=1651–1654|issn=1462-0324|doi=10.1093/rheumatology/ken371}}</ref><ref name="PapaDoglio2017">{{cite journal|last1=Papa|first1=Riccardo|last2=Doglio|first2=Matteo|last3=Lachmann|first3=Helen J.|last4=Ozen|first4=Seza|last5=Frenkel|first5=Joost|last6=Simon|first6=Anna|last7=Neven|first7=Bénédicte|last8=Kuemmerle-Deschner|first8=Jasmin|last9=Ozgodan|first9=Huri|last10=Caorsi|first10=Roberta|last11=Federici|first11=Silvia|last12=Finetti|first12=Martina|last13=Trachana|first13=Maria|last14=Brunner|first14=Jurgen|last15=Bezrodnik|first15=Liliana|last16=Pinedo Gago|first16=Mari Carmen|last17=Maggio|first17=Maria Cristina|last18=Tsitsami|first18=Elena|last19=Al Suwairi|first19=Wafaa|last20=Espada|first20=Graciela|last21=Shcherbina|first21=Anna|last22=Aksu|first22=Guzide|last23=Ruperto|first23=Nicolino|last24=Martini|first24=Alberto|last25=Ceccherini|first25=Isabella|last26=Gattorno|first26=Marco|title=A web-based collection of genotype-phenotype associations in hereditary recurrent fevers from the Eurofever registry|journal=Orphanet Journal of Rare Diseases|volume=12|issue=1|year=2017|issn=1750-1172|doi=10.1186/s13023-017-0720-3}}</ref>
* Other possible [[risk factors]] include:
* Other possible [[risk factors]] include:
** [[Obesity]]
**[[Obesity]]
** Aging
** Aging
** SAA1 gene alleles
** SAA1 [[gene]] alleles
** Monogenic [[periodic fever syndromes]], such as:
** Monogenic [[Periodic fever syndrome|periodic fever syndromes]], such as:
*** [[FMF]]
***[[FMF]]
*** [[TNF Receptor associated periodic syndrome]] ([[TRAPS]])
***[[TNF receptor associated periodic syndrome|TNF Receptor associated periodic syndrome]] ([[TRAPS]])
*** Cryopyrin-associated periodic fever syndrome
***[[CAPS|Cryopyrin-associated periodic fever syndrome]]
*** Mevalonate kinase deficiency
***[[Mevalonate kinase deficiency]]
** Tuberculosis
**[[Tuberculosis]]
** Leprosy
**[[Leprosy]]
** Whipple Disease
** Whipple Disease
** Osteomyelitis
**[[Osteomyelitis]]
** Chronic pyelonephritis
**[[Chronic pyelonephritis]]
** Subacute bacterial endocarditis
**[[Subacute bacterial endocarditis]]
** Chronic cutaneous ulcers
** Chronic cutaneous ulcers
* Conditions Predisposing to Chronic Infections include:
*[[Conditions]] Predisposing to chronic [[infections]] include:
** Cystic fibrosis
**[[Cystic fibrosis]]
** Bronchiectasis
**[[Bronchiectasis]]
** Kartagener syndrome
**[[Kartagener syndrome]]
** Epidermolysis bullosa
**[[Epidermolysis bullosa]]
** Injected drug abuse
** Injected drug abuse
** Jejuno-ileal bypass
**[[Jejuno-ileal bypass]]
** Paraplegia
**[[Paraplegia]]
** Sickle cell anemia
**[[Sickle cell anemia]]
** Immunodeficiency
**[[Immunodeficiency]]
** Common variable immunodeficiency
**[[Common variable immunodeficiency]]
** Cyclic neutropenia
**[[Cyclic neutropenia]]
** Hyperimmunoglobulin M syndrome
**[[Hyperimmunoglobulin M syndrome]]
** Hypogammaglobulinemia
**[[Hypogammaglobulinemia]]
** Sex-linked agammaglobulinemia
** Sex-linked agammaglobulinemia
** Human immunodeficiency virus/AIDS
**[[Human Immunodeficiency Virus|Human immunodeficiency virus]]/[[AIDS]]
** Neoplasia
**[[Neoplasia]]
** Adenocarcinoma
**[[Adenocarcinoma]]
Basal cell carcinoma
**[[Basal cell carcinoma]]
Carcinoid tumor
**[[Carcinoid tumors|Carcinoid tumor]]
Castleman disease
**[[Castleman disease]]
Gastrointestinal stromal tumor
**[[Gastrointestinal stromal tumor]]
Hairy cell leukemia
**[[Hairy cell leukemia]]
Hepatic adenoma
**[[Hepatic adenoma]]
Hodgkin disease
**[[Hodgkin disease]]
** Mesothelioma
**[[Mesothelioma]]
** Renal cell carcinoma
**[[Renal cell carcinoma]]
** Sarcoma
**[[Sarcoma]]
** Inflammatory Arthritis
** Inflammatory Arthritis
** Adult-onset Still disease
** Adult-onset Still disease
** Ankylosing spondylitis
**[[Ankylosing spondylitis]]
** Juvenile idiopathic arthritis
**[[Juvenile idiopathic arthritis]]
** Psoriatic arthropathy
**[[Psoriatic arthropathy]]
** Reiter syndrome
**[[Reiter's syndrome|Reiter syndrome]]
** Rheumatoid arthritis
**[[Rheumatoid arthritis]]
** Gout
**[[Gout]]
** Systemic Vasculitis
** Systemic [[Vasculitis]]
** Antineutrophil cytoplasmic antibody-associated vasculitis
** Antineutrophil cytoplasmic antibody-associated vasculitis
** Behcet disease
**[[Behcet disease]]
** Giant cell arteritis
**[[Giant cell arteritis]]
** Polyarteritis nodosa
**[[Polyarteritis nodosa]]
** Polymyalgia rheumatica
**[[Polymyalgia rheumatica]]
** Systemic lupus erythematosus
**[[Systemic lupus erythematosus]]
Takayasu arteritis
**[[Takayasu arteritis]]
** Inflammatory Bowel Disease
**[[Inflammatory Bowel Disease]]
** Ulcerative colitis
**[[Ulcerative colitis]]
** Crohn diseases
**[[Crohn disease]]
* Others include:
* Others include:
** Atrial myxoma
**[[Atrial myxoma]]
** Inflammatory abdominal aortic aneurism
** Inflammatory abdominal aortic aneurism
** Retroperitoneal fibrosis
**[[Retroperitoneal fibrosis]]
** SAPHO (synovitis, acne, pustulosis, hyperostosis, and osteitis) syndrome
**[[SAPHO syndrome|SAPHO (synovitis, acne, pustulosis, hyperostosis, and osteitis) syndrome]]
** Sarcoidosis
**[[Sarcoidosis]]
** Sinus histiocytosis with massive lymphadenopathy
**[[Sinus histiocytosis with massive lymphadenopathy]]
 
==References==
==References==
{{Reflist|2}}
{{Reflist|2}}

Latest revision as of 14:00, 30 October 2019

Secondary amyloidosis Microchapters

Home

Patient Information

Overview

Historical Perspective

Classification

Pathophysiology

Causes

Differentiating Secondary amyloidosis from other Diseases

Epidemiology and Demographics

Risk Factors

Screening

Natural History, Complications and Prognosis

Diagnosis

Diagnostic Study of Choice

History and Symptoms

Physical Examination

Laboratory Findings

Electrocardiogram

X-ray

Echocardiography and Ultrasound

CT scan

MRI

Other Imaging Findings

Other Diagnostic Studies

Treatment

Medical Therapy

Surgery

Primary Prevention

Secondary Prevention

Case Studies

Case #1

Secondary amyloidosis risk factors On the Web

Most recent articles

Most cited articles

Review articles

CME Programs

Powerpoint slides

Images

American Roentgen Ray Society Images of Secondary amyloidosis risk factors

All Images
X-rays
Echo & Ultrasound
CT Images
MRI

Ongoing Trials at Clinical Trials.gov

US National Guidelines Clearinghouse

NICE Guidance

FDA on Secondary amyloidosis risk factors

CDC on Secondary amyloidosis risk factors

Secondary amyloidosis risk factors in the news

Blogs on Secondary amyloidosis risk factors

Directions to Hospitals Treating Psoriasis

Risk calculators and risk factors for Secondary amyloidosis risk factors

Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] Associate Editor(s)-in-Chief: Sahar Memar Montazerin, M.D.[2]

Overview

The most potent risk factor in the development of secondary amyloidosis is a persistent inflammatory disorders. Chronic infections and inflammatory arthritis are among the most common risk factors.

Risk Factors

References

  1. ↑ Koivuniemi, Riitta; Paimela, Leena; Suomalainen, Risto; Törnroth, Tom; Leirisalo-Repo, Marjatta (2009). "Amyloidosis is frequently undetected in patients with rheumatoid arthritis". Amyloid. 15 (4): 262–268. doi:10.1080/13506120802524676. ISSN 1350-6129.
  2. ↑ Blank, Norbert; Hegenbart, Ute; Dietrich, Sascha; Brune, Maik; Beimler, Jörg; Röcken, Christoph; Müller-Tidow, Carsten; Lorenz, Hanns-Martin; Schönland, Stefan O. (2018). "Obesity is a significant susceptibility factor for idiopathic AA amyloidosis". Amyloid. 25 (1): 37–45. doi:10.1080/13506129.2018.1429391. ISSN 1350-6129.
  3. ↑ van der Hilst, J. C. H.; Yamada, T.; Op den Camp, H. J. M.; van der Meer, J. W. M.; Drenth, J. P. H.; Simon, A. (2008). "Increased susceptibility of serum amyloid A 1.1 to degradation by MMP-1: potential explanation for higher risk of type AA amyloidosis". Rheumatology. 47 (11): 1651–1654. doi:10.1093/rheumatology/ken371. ISSN 1462-0324.
  4. ↑ Papa, Riccardo; Doglio, Matteo; Lachmann, Helen J.; Ozen, Seza; Frenkel, Joost; Simon, Anna; Neven, Bénédicte; Kuemmerle-Deschner, Jasmin; Ozgodan, Huri; Caorsi, Roberta; Federici, Silvia; Finetti, Martina; Trachana, Maria; Brunner, Jurgen; Bezrodnik, Liliana; Pinedo Gago, Mari Carmen; Maggio, Maria Cristina; Tsitsami, Elena; Al Suwairi, Wafaa; Espada, Graciela; Shcherbina, Anna; Aksu, Guzide; Ruperto, Nicolino; Martini, Alberto; Ceccherini, Isabella; Gattorno, Marco (2017). "A web-based collection of genotype-phenotype associations in hereditary recurrent fevers from the Eurofever registry". Orphanet Journal of Rare Diseases. 12 (1). doi:10.1186/s13023-017-0720-3. ISSN 1750-1172.
Retrieved from "https://www.wikidoc.org/index.php?title=Secondary_amyloidosis_risk_factors&oldid=1587200"