Cardiomyopathy physical examination: Difference between revisions
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{{Cardiomyopathy}} {{CMG}} | {{Cardiomyopathy}} | ||
{{CMG}}; {{AE}} [[User:Lina Ya'qoub|Lina Ya'qoub, MD]]; {{EdzelCo}} | |||
==Overview== | ==Overview== | ||
A thorough physical examination can help in diagnosing cardiomyopathy. Particular attention should be given to the cardiac exam, the lung exam, the extremities for [[edema]], and in assessing for other signs of [[heart failure]] such as [[jugular venous distension]]. | A thorough physical examination can help in diagnosing cardiomyopathy. Particular attention should be given to the cardiac exam, the lung exam, the extremities for [[edema]], and in assessing for other signs of [[heart failure]] such as [[jugular venous distension]]. | ||
==Physical Examination== | ==Physical Examination== | ||
The most important physical exam aspects of cardiomyopathies include:<ref name="pmid24976920">{{cite journal |vauthors=Sisakian H |title=Cardiomyopathies: Evolution of pathogenesis concepts and potential for new therapies |journal=World J Cardiol |volume=6 |issue=6 |pages=478–94 |date=June 2014 |pmid=24976920 |doi=10.4330/wjc.v6.i6.478 |url=}}</ref> | |||
=== '''Vital signs''' === | |||
* It is always very important to measure vital signs in all patients with any cause of cardiomyopathy. | |||
=== '''Neck''' === | |||
* [[jugular venous distension]] might be noted when the right atrial pressures are elevated. | |||
* A bifid carotid upstroke might be noted in HCM. | |||
===Heart=== | ===Heart=== | ||
* Apical impulse might be displaced in dilated cardiomyopathy or forceful in [[HCM]]. | |||
* A systolic murmur might be heard at the apex, radiating to the axilla, which might indicate LV outflow obstruction in [[HCM]] or the presence of mitral regurgitation. | |||
* The systolic murmur of [[HCM]] is increased by [[valsalva]] maneuver or standing. | |||
===Lungs=== | ===Lungs=== | ||
Crackles in the lungs may be a sign of [[heart failure]], which often develops in the later stages of cardiomyopathy. | * Crackles in the lungs may be a sign of [[heart failure]], which often develops in the later stages of cardiomyopathy. | ||
===Extremities=== | ===Extremities=== | ||
[[Edema]] of the ankles, feet, legs | * [[Edema]] of the ankles, feet, legs and abdomen. | ||
==References== | ==References== | ||
Latest revision as of 03:27, 27 October 2023
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Cardiomyopathy Microchapters |
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Diagnosis |
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Treatment |
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Guidelines |
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2020 AHA/ACC Guideline for the Diagnosis and Treatment of Patients With Hypertrophic Cardiomyopathy |
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Case Studies |
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Cardiomyopathy physical examination On the Web |
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American Roentgen Ray Society Images of Cardiomyopathy physical examination |
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Risk calculators and risk factors for Cardiomyopathy physical examination |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Lina Ya'qoub, MD; Edzel Lorraine Co, DMD, MD[2]
Overview
A thorough physical examination can help in diagnosing cardiomyopathy. Particular attention should be given to the cardiac exam, the lung exam, the extremities for edema, and in assessing for other signs of heart failure such as jugular venous distension.
Physical Examination
The most important physical exam aspects of cardiomyopathies include:[1]
Vital signs
- It is always very important to measure vital signs in all patients with any cause of cardiomyopathy.
Neck
- jugular venous distension might be noted when the right atrial pressures are elevated.
- A bifid carotid upstroke might be noted in HCM.
Heart
- Apical impulse might be displaced in dilated cardiomyopathy or forceful in HCM.
- A systolic murmur might be heard at the apex, radiating to the axilla, which might indicate LV outflow obstruction in HCM or the presence of mitral regurgitation.
- The systolic murmur of HCM is increased by valsalva maneuver or standing.
Lungs
- Crackles in the lungs may be a sign of heart failure, which often develops in the later stages of cardiomyopathy.
Extremities
- Edema of the ankles, feet, legs and abdomen.
References
- ↑ Sisakian H (June 2014). "Cardiomyopathies: Evolution of pathogenesis concepts and potential for new therapies". World J Cardiol. 6 (6): 478–94. doi:10.4330/wjc.v6.i6.478. PMID 24976920.