Thrombosis risk stratification: Difference between revisions
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{{Thrombosis}} | {{Thrombosis}} | ||
{{CMG}} | {{CMG}} {{AE}} {{VE}} | ||
==Overview== | ==Overview== | ||
Presence of a risk factor for thrombosis has an impact on the treatment of the patient. Short term anti-coagulation can be successful in the presence of a transient risk factor. Long term anti-coagulation is required in case of a persistent risk factor. | Presence of a [[thrombophilia|risk factor for thrombosis]] has an impact on the treatment of the patient. Short term anti-coagulation can be successful in the presence of a transient risk factor. Long term anti-coagulation is required in case of a persistent risk factor. | ||
==Risk Factors== | ==Risk Factors== | ||
The Thrombosis formation is a phenomenon that can be elicited by various conditions, all of which take effect by means of mechanisms of [[Virchow's triad]]. Blood stasis, endothelial defect or [[hypercoagulability]]. | |||
*General | *General | ||
**Older age | **Older age | ||
Latest revision as of 09:05, 29 January 2019
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Thrombosis Microchapters |
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Site of Thrombosis |
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Differentiating Thrombosis from other Diseases |
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Diagnosis |
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Treatment |
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Thrombosis risk stratification On the Web |
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Risk calculators and risk factors for Thrombosis risk stratification |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] Associate Editor(s)-in-Chief: Vahid Eidkhani, M.D.
Overview
Presence of a risk factor for thrombosis has an impact on the treatment of the patient. Short term anti-coagulation can be successful in the presence of a transient risk factor. Long term anti-coagulation is required in case of a persistent risk factor.
Risk Factors
The Thrombosis formation is a phenomenon that can be elicited by various conditions, all of which take effect by means of mechanisms of Virchow's triad. Blood stasis, endothelial defect or hypercoagulability.
- Medical
- Familial
- Antithrombin III deficiency
- Protein C deficiency/Protein S deficiency
- APC resistance (Factor V Leiden)
- Dysfibrogenemia
- Hypoplasminogenemia
- Familial homocysteinemia