Dyspepsia classification: Difference between revisions
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==Classification== | ==Classification== | ||
===Ulcer Dyspepsia=== | ===Ulcer Dyspepsia=== | ||
Ulcer dyspepsia accounts to 20-30 % of all dyspepsia cases and is caused by [[peptic ulcer disease]], [[GERD]]. Structural disease of the gastrointestinal tract exists in ulcer dyspepsia and hence endoscopy is abnormal. | * Ulcer dyspepsia accounts to 20-30 % of all dyspepsia cases and is caused by [[peptic ulcer disease]], [[GERD]]. | ||
* Structural disease of the gastrointestinal tract exists in ulcer dyspepsia and hence endoscopy is abnormal. | |||
===Non-Ulcer Dyspepsia=== | ===Non-Ulcer Dyspepsia=== | ||
Non-ulcer dyspepsia, also called functional dyspepsia (FD), is defined by the Rome III criteria as symptoms of epigastric pain or discomfort (prevalence in FD of 89-90%), postprandial fullness (75-88%), and early satiety (50-82%) within the last 3 months with symptom onset at least 6 months earlier. Patients cannot have any evidence of structural disease to explain symptoms and predominant symptoms of gastroesophageal reflux are exclusionary.<ref>{{cite journal |author=Lacy BE, Talley NJ, Locke GR, ''et al.'' |title=Review article: current treatment options and management of functional dyspepsia |journal=Aliment. Pharmacol. Ther. |volume=36 |issue=1 |pages=3–15 |year=2012 |month=July |pmid=22591037 |doi=10.1111/j.1365-2036.2012.05128.x |url=}}</ref> | * Non-[[ulcer]] dyspepsia, also called functional dyspepsia (FD), is defined by the [[Rome III criteria]] as symptoms of [[epigastric pain]] or [[discomfort]] ([[prevalence]] in FD of 89-90%), [[postprandial]] fullness (75-88%), and early [[satiety]] (50-82%) within the last 3 months with symptom onset at least 6 months earlier. | ||
* Patients cannot have any evidence of structural disease to explain [[symptoms]] and predominant symptoms of [[gastroesophageal reflux]] are exclusionary.<ref>{{cite journal |author=Lacy BE, Talley NJ, Locke GR, ''et al.'' |title=Review article: current treatment options and management of functional dyspepsia |journal=Aliment. Pharmacol. Ther. |volume=36 |issue=1 |pages=3–15 |year=2012 |month=July |pmid=22591037 |doi=10.1111/j.1365-2036.2012.05128.x |url=}}</ref> | |||
Causes of functional dyspepsia are not clear but researchers have focused on the following factors: | * Causes of functional dyspepsia are not clear but researchers have focused on the following factors: | ||
*Gastric motor function | **[[Gastric]] motor function | ||
*Visceral sensitivity | **[[Visceral]] [[sensitivity]] | ||
*Helicobacter pylori infection | **[[Helicobacter pylori infection]] | ||
*Psychosocial factors | **[[Psychosocial]] factors | ||
==References== | ==References== | ||
Revision as of 15:51, 29 January 2018
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Overview
Dyspepsia is broadly classified into two major types: ulcer and non-ulcer dyspepsia. The latter is also known as functional dyspepsia.
Classification
Ulcer Dyspepsia
- Ulcer dyspepsia accounts to 20-30 % of all dyspepsia cases and is caused by peptic ulcer disease, GERD.
- Structural disease of the gastrointestinal tract exists in ulcer dyspepsia and hence endoscopy is abnormal.
Non-Ulcer Dyspepsia
- Non-ulcer dyspepsia, also called functional dyspepsia (FD), is defined by the Rome III criteria as symptoms of epigastric pain or discomfort (prevalence in FD of 89-90%), postprandial fullness (75-88%), and early satiety (50-82%) within the last 3 months with symptom onset at least 6 months earlier.
- Patients cannot have any evidence of structural disease to explain symptoms and predominant symptoms of gastroesophageal reflux are exclusionary.[1]
- Causes of functional dyspepsia are not clear but researchers have focused on the following factors:
- Gastric motor function
- Visceral sensitivity
- Helicobacter pylori infection
- Psychosocial factors