Oral cancer primary prevention: Difference between revisions
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===Human papillomavirus (HPV) Infection=== | ===Human papillomavirus (HPV) Infection=== | ||
Vaccination against HPV 16 and 18 has been shown to prevent more than 90% of oral HPV 16/18 infections within 4 years of vaccination. Given the relatively recent onset of vaccination adoption and the age at which individuals are vaccinated, there is not yet evidence that [[vaccination]] at a young age will lead to a substantially reduced risk of HPV-associated [[oropharyngeal]] cancer later in life. In addition, no data are available to examine whether [[incidence]] or [[mortality]] would be reduced if vaccination occurred at an age closer to that at which oropharyngeal cancers tend to present.<ref>{{Cite web | title =NIH Prevention for lip and oral cancer treatment factors| url =http://www.cancer.gov/types/head-and-neck/hp/oral-prevention-pdq}}</ref> | |||
==References== | ==References== | ||
Revision as of 13:33, 14 September 2015
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Oral cancer Microchapters |
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Diagnosis |
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Treatment |
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Case Studies |
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Oral cancer primary prevention On the Web |
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American Roentgen Ray Society Images of Oral cancer primary prevention |
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Risk calculators and risk factors for Oral cancer primary prevention |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]Associate Editor(s)-in-Chief: Simrat Sarai, M.D. [2]
Overview
Effective measures for the primary prevention of oral cancer include tobacco cessation, alcohol cessation, eating variety of fruits and vegetables, and avoiding excessive sun exposure.
Primary Prevention
Tobacco cessation
Based on solid evidence, cessation of exposure to tobacco (e.g., cigarettes, pipes, cigars, and smokeless tobacco) leads to a decrease in the risk of cancer of the oral cavity and oropharynx.
Cessation of alcohol consumption
Based on fair evidence, cessation of alcohol consumption leads to a decrease in oral cavity cancer, but not until approximately 10 years after cessation. For cancer of the oropharynx, reduction in risk does not occur until approximately 20 years after cessation.
Human papillomavirus (HPV) Infection
Vaccination against HPV 16 and 18 has been shown to prevent more than 90% of oral HPV 16/18 infections within 4 years of vaccination. Given the relatively recent onset of vaccination adoption and the age at which individuals are vaccinated, there is not yet evidence that vaccination at a young age will lead to a substantially reduced risk of HPV-associated oropharyngeal cancer later in life. In addition, no data are available to examine whether incidence or mortality would be reduced if vaccination occurred at an age closer to that at which oropharyngeal cancers tend to present.[1]