Gliomatosis cerebri pathophysiology: Difference between revisions
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===Associated Conditions=== | ===Associated Conditions=== | ||
Gliomatosis cerebri may be associated with: | Gliomatosis cerebri may be associated with:<ref name="pmid21837541">{{cite journal| author=Buis DR, van der Valk P, De Witt Hamer PC| title=Subcutaneous tumor seeding after biopsy in gliomatosis cerebri. | journal=J Neurooncol | year= 2012 | volume= 106 | issue= 2 | pages= 431-5 | pmid=21837541 | doi=10.1007/s11060-011-0678-2 | pmc=PMC3230756 | url=http://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=21837541 }} </ref> | ||
*[[Neurofibromatosis type 1]] | *[[Neurofibromatosis type 1]] | ||
Revision as of 13:39, 25 September 2015
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]Associate Editor(s)-in-Chief: Sujit Routray, M.D. [2]
Overview
Pathophysiology
Pathogenesis
- Gliomatosis cerebri may be classified into primary (de novo) or secondary, with the latter as a result from the spreading of a more focal glioma.
- According to WHO classification of brain tumors, gliomatosis cerebri is classified into grade 2 or grade 3 tumors.
Associated Conditions
Gliomatosis cerebri may be associated with:[1]
Gross Pathology
On gross pathology, gliomatosis cerebri is characterized by:
- Diffuse, usually astrocytic growth pattern
- Involves at least three cerebral lobes
- Bilateral involvement of the cerebral hemispheres, deep gray matter, brainstem, or cerebellum
References
- ↑ Buis DR, van der Valk P, De Witt Hamer PC (2012). "Subcutaneous tumor seeding after biopsy in gliomatosis cerebri". J Neurooncol. 106 (2): 431–5. doi:10.1007/s11060-011-0678-2. PMC 3230756. PMID 21837541.