Hyporeflexia: Difference between revisions
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| '''Nutritional / Metabolic''' | | '''Nutritional / Metabolic''' | ||
|bgcolor="Beige"| [[3-methylglutaconic aciduria type 4]], | |bgcolor="Beige"| [[3-methylglutaconic aciduria type 4]], [[congenital disorder of glycosylation]], [[cytochrome c oxidase deficiency]], [[folate deficiency]], [[hypermagnesaemia]], [[hypokalemia]], [[hypokalemic periodic paralysis]], [[Krabbe disease]], [[Leigh syndrome]], [[Mcleod phenotype]], [[NADH CoQ reductase deficiency]], [[mitochondrial acetoacetyl-CoA thiolase deficiency]], navajo neurohepatopathy, non-ketotic hyperglycemia, Pena-shokeir syndrome type 2, [[tabes dorsalis]], [[vitamin E deficiency]] | ||
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[[Krabbe disease | |||
[[Leigh syndrome | |||
[[Mcleod phenotype]], | |||
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Revision as of 17:14, 15 November 2015
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] Carlos A Lopez, M.D. [2]
Overview
Causes
Life Threatening Causes
Life-threatening causes include conditions which may result in death or permanent disability within 24 hours if left untreated.
Common Causes
Causes by Organ System
Causes in Alphabetical Order
References
References
See also
- Areflexia, the absence of reflexes, often it is caused by spinal shock.
- Hyperreflexia, exaggerated reflexes.