Thrombosis risk stratification: Difference between revisions
Jump to navigation
Jump to search
No edit summary |
No edit summary |
||
| Line 7: | Line 7: | ||
==Risk Factors== | ==Risk Factors== | ||
The Thrombosis formation is a phenomenon that can be elicited by various conditions, all of which take effect by means of mechanisms of [[Virchow's triad]]. Blood stasis, endothelial defect or [[hypercoagulability]]. | |||
*General | *General | ||
**Older age | **Older age | ||
Revision as of 08:22, 27 August 2018
|
Thrombosis Microchapters |
|
Site of Thrombosis |
|---|
|
Differentiating Thrombosis from other Diseases |
|
Diagnosis |
|
Treatment |
|
Thrombosis risk stratification On the Web |
|
Risk calculators and risk factors for Thrombosis risk stratification |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Overview
Presence of a risk factor for thrombosis has an impact on the treatment of the patient. Short term anti-coagulation can be successful in the presence of a transient risk factor. Long term anti-coagulation is required in case of a persistent risk factor.
Risk Factors
The Thrombosis formation is a phenomenon that can be elicited by various conditions, all of which take effect by means of mechanisms of Virchow's triad. Blood stasis, endothelial defect or hypercoagulability.
- Medical
- Familial
- Antithrombin III deficiency
- Protein C deficiency/Protein S deficiency
- APC resistance (Factor V Leiden)
- Dysfibrogenemia
- Hypoplasminogenemia
- Familial homocysteinemia