Tricuspid stenosis natural history, complications and prognosis: Difference between revisions
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==Overview== | ==Overview== | ||
The natural course of tricuspid stenosis is not well defined and it is extremely rare for TS to occur in isolation.<ref name="pmid9665226">{{cite journal| author=Roguin A, Rinkevich D, Milo S, Markiewicz W, Reisner SA| title=Long-term follow-up of patients with severe rheumatic tricuspid stenosis. | journal=Am Heart J | year= 1998 | volume= 136 | issue= 1 | pages= 103-8 | pmid=9665226 | doi= | pmc= | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=9665226 }} </ref> The most common cause of TS is rheumatic heart disease and it is usually associated with coexisting mitral valve and/or aortic valve abnormality.<ref name="pmid9665226">{{cite journal| author=Roguin A, Rinkevich D, Milo S, Markiewicz W, Reisner SA| title=Long-term follow-up of patients with severe rheumatic tricuspid stenosis. | journal=Am Heart J | year= 1998 | volume= 136 | issue= 1 | pages= 103-8 | pmid=9665226 | doi= | pmc= | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=9665226 }} </ref> TS of rheumatic etiology usually occurs with tricuspid regurgitation. Tricuspid stenosis takes years to develop, with the exception of congenital complications of tricuspid stenosis include [[heart failure]], [[liver failure]], and [[stroke]].<ref name="pmid15786615">{{cite journal| author=Diaof M, Ba SA, Kane A, Sarr M, Diop IB, Diouf SM| title=[Tricuspid valve stenosis. A prospective study of 35 cases]. | journal=Dakar Med | year= 2004 | volume= 49 | issue= 2 | pages= 96-100 | pmid=15786615 | doi= | pmc= | url=http://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=15786615 }} </ref> | |||
==Complication== | ==Complication== | ||
The complications | The following complications may occur from tricuspid stenosis: | ||
* Right atrial enlargement | * Right atrial enlargement | ||
* Atrial fibrillation | * Atrial fibrillation | ||
* | * Heart failure | ||
* Cerebrovascular accidents | |||
==Prognosis== | ==Prognosis== | ||
With medical intervention, severe tricuspid stenosis appears well tolerated over several years of follow-up.<ref name="pmid9665226">{{cite journal| author=Roguin A, Rinkevich D, Milo S, Markiewicz W, Reisner SA| title=Long-term follow-up of patients with severe rheumatic tricuspid stenosis. | journal=Am Heart J | year= 1998 | volume= 136 | issue= 1 | pages= 103-8 | pmid=9665226 | doi= | pmc= | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=9665226 }} </ref> | |||
==References== | ==References== | ||
{{Reflist|2}} | {{Reflist|2}} |
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Overview
The natural course of tricuspid stenosis is not well defined and it is extremely rare for TS to occur in isolation.[1] The most common cause of TS is rheumatic heart disease and it is usually associated with coexisting mitral valve and/or aortic valve abnormality.[1] TS of rheumatic etiology usually occurs with tricuspid regurgitation. Tricuspid stenosis takes years to develop, with the exception of congenital complications of tricuspid stenosis include heart failure, liver failure, and stroke.[2]
Complication
The following complications may occur from tricuspid stenosis:
- Right atrial enlargement
- Atrial fibrillation
- Heart failure
- Cerebrovascular accidents
Prognosis
With medical intervention, severe tricuspid stenosis appears well tolerated over several years of follow-up.[1]
References
- ↑ 1.0 1.1 1.2 Roguin A, Rinkevich D, Milo S, Markiewicz W, Reisner SA (1998). "Long-term follow-up of patients with severe rheumatic tricuspid stenosis". Am Heart J. 136 (1): 103–8. PMID 9665226 Check
|pmid=
value (help). zero width space character in|pmid=
at position 8 (help) - ↑ Diaof M, Ba SA, Kane A, Sarr M, Diop IB, Diouf SM (2004). "[Tricuspid valve stenosis. A prospective study of 35 cases]". Dakar Med. 49 (2): 96–100. PMID 15786615.