Renal agenesis overview: Difference between revisions
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It can be associated with ''[[RET proto-oncogene|RET]]'' or ''[[UPK3A]]''.<ref>{{OMIM|191830}}</ref> | It can be associated with ''[[RET proto-oncogene|RET]]'' or ''[[UPK3A]]''.<ref>{{OMIM|191830}}</ref> | ||
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== Epidemiology and Demographics == | |||
The general [[incidence]] of unilateral renal agenesis (URA) has been reported to be approximately 1 in 2031 individuals. The [[incidence]] of bilateral renal agenesis (BRA) is approximately 1 in every 3000 pregnancies. [[Male|Males]] are more commonly affected by unilateral renal agenesis (URA) than [[Female|females]]. The [[mortality rate]] of bilateral renal agenesis (BRA) without [[Prenatal development|prenatal]] therapy is 100%. | |||
==References== | ==References== |
Revision as of 12:15, 22 July 2020
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Overview
Renal agenesis is the absence of one (unilateral) or both (bilateral) kidneys at birth. Renal agenesis is a medical condition in which one (unilateral) or both (bilateral) fetal kidneys fail to develop.
It can be associated with RET or UPK3A.[1]
Epidemiology and Demographics
The general incidence of unilateral renal agenesis (URA) has been reported to be approximately 1 in 2031 individuals. The incidence of bilateral renal agenesis (BRA) is approximately 1 in every 3000 pregnancies. Males are more commonly affected by unilateral renal agenesis (URA) than females. The mortality rate of bilateral renal agenesis (BRA) without prenatal therapy is 100%.