Hypoaldosteronism secondary prevention: Difference between revisions
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==Overview== | ==Overview== | ||
Effective measures for the secondary prevention of hypoaldosteronism include liberal salt intake of 4gm/day (to increase plasma sodium concentration), decreasing potassium intake and avoidance of drugs that affects renin angiotensin aldosterone system (RAAS) such as ACE inhibitors, ARBs, potassium sparing diuretics and β-Adrenergic receptor blockers. | |||
Effective measures for the secondary prevention of | |||
==Secondary Prevention== | ==Secondary Prevention== | ||
Revision as of 14:39, 22 August 2017
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Hypoaldosteronism Microchapters |
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Hypoaldosteronism secondary prevention On the Web |
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief:
Overview
Effective measures for the secondary prevention of hypoaldosteronism include liberal salt intake of 4gm/day (to increase plasma sodium concentration), decreasing potassium intake and avoidance of drugs that affects renin angiotensin aldosterone system (RAAS) such as ACE inhibitors, ARBs, potassium sparing diuretics and β-Adrenergic receptor blockers.
Secondary Prevention
- Effective measures for the secondary prevention of hypoaldosteronism include:
- Low potassium intake
- Salt intake of 4gm/day
- Avoid drugs affecting the renin angiotensin aldosterone system (RAAS) such as:
- ACE inhibitors
- Angiotensinogen receptor blocker
- Potassium sparing diuretics
- β-Adrenergic receptor blockers