Sandbox: sadaf: Difference between revisions
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| bgcolor="LightSteelBlue" | '''Chemical/Poisoning''' | | bgcolor="LightSteelBlue" | '''Chemical/Poisoning''' | ||
| bgcolor="Beige" | | | bgcolor="Beige" | [[St John's wort]] | ||
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| '''Drug Side Effect''' | | '''Drug Side Effect''' | ||
| bgcolor="Beige" | | | bgcolor="Beige" | [[Glucocorticoid]], [[Highly Active Antiretroviral Therapy]] ([[HAART]]), [[Phenobarbital]], [[Phenytoin]], [[Primidone]], [[Rifampin]], [[Valproate]] | ||
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| '''Endocrine''' | | '''Endocrine''' | ||
| bgcolor="Beige" | | | bgcolor="Beige" | [[Autosomal-dominant hypophosphatemic rickets]], [[Hyperthyroidism]], [[Primary hyperparathyroidism]], [[Type I hereditary vitamin D–dependent rickets]], [[Type II hereditary vitamin D–dependent rickets]], [[Vitamin D-resistant rickets]], [[X-linked familial hypophosphatemia]] | ||
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| '''Gastroenterologic''' | | '''Gastroenterologic''' | ||
| bgcolor="Beige" | | | bgcolor="Beige" | [[Celiac disease]], [[Crohn disease]], [[Cystic fibrosis]], [[Liver failure]], Post [[gastric bypass surgery]], [[Short bowel syndrome]], [[Whipple disease]] | ||
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| '''Genetic''' | | '''Genetic''' | ||
| bgcolor="Beige" | | | bgcolor="Beige" | [[Autosomal-dominant hypophosphatemic rickets]], [[Type I hereditary vitamin D–dependent rickets]], [[Type II hereditary vitamin D–dependent rickets]], [[Vitamin D-resistant rickets]], [[X-linked familial hypophosphatemia]] | ||
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| '''Infectious Disease''' | | '''Infectious Disease''' | ||
| bgcolor="Beige" | | | bgcolor="Beige" | [[Tuberculosis]] | ||
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|- bgcolor="LightSteelBlue" | |- bgcolor="LightSteelBlue" | ||
| '''Musculoskeletal/Orthopedic''' | | '''Musculoskeletal/Orthopedic''' | ||
| bgcolor="Beige" | | | bgcolor="Beige" | [[Autosomal-dominant hypophosphatemic rickets]], [[Type I hereditary vitamin D–dependent rickets]], [[Type II hereditary vitamin D–dependent rickets]], [[Vitamin D-resistant rickets]], [[X-linked familial hypophosphatemia]] | ||
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| '''Nutritional/Metabolic''' | | '''Nutritional/Metabolic''' | ||
| bgcolor="Beige" | | | bgcolor="Beige" | Inadequate [[vitamin D]] dietary intake | ||
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| '''Renal/Electrolyte''' | | '''Renal/Electrolyte''' | ||
| bgcolor="Beige" | | | bgcolor="Beige" | [[Chronic kidney disease]], [[Nephrotic syndrome]] | ||
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|- bgcolor="LightSteelBlue" | |- bgcolor="LightSteelBlue" | ||
| '''Rheumatology/Immunology/Allergy''' | | '''Rheumatology/Immunology/Allergy''' | ||
| bgcolor="Beige" | | | bgcolor="Beige" | [[Sarcoidosis]] | ||
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Revision as of 22:16, 25 August 2017
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Vitamin D deficiency Microchapters |
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Diagnosis |
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Treatment |
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Case Studies |
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Sandbox: sadaf On the Web |
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American Roentgen Ray Society Images of Sandbox: sadaf |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] Associate Editor(s)-in-Chief: Sadaf Sharfaei M.D.[2]
Overview
Common causes of vitamin D deficiency are inadequate vitamin D skin production, less dietary vitamin D intake and impaired absorption. Other causes of vitamin D deficiency include vitamin D loss, abnormal metabolism, resistance to vitamin D and medication. [1]
Causes
Vitamin D deficiency may be caused by: [1]
- Inadequate vitamin D skin production
- Inadequate vitamin D dietary intake
- Vitamin D loss
- Malabsorption
- Impaired metabolism
- Resistance to Vitamin D
- Medication
Common causes
- Antiepileptic medication [2]
- Glucocorticoid [3]
- Inadequate sun exposure including dark skin [4] and clothing [5]
- Inadequate vitamin D dietary intake
Less common cause
- Autosomal-dominant hypophosphatemic rickets
- Celiac disease
- Chronic kidney disease: impaired renal 1α-hydroxylation [6]
- Crohn disease [7][8]
- Cystic fibrosis
- Highly active antiretroviral therapy (HAART)
- Hyperthyroidism
- Impaired enterohepatic circulation
- Nephrotic syndrome
- Post gastric bypass surgery
- Primary hyperparathyroidism
- Rifampin
- Sarcoidosis
- Severe liver failure: impaired hepatic 25-hydroxylation
- Short bowel syndrome
- St John's wort
- Tuberculosis
- Type I hereditary vitamin D–dependent rickets
- Type II hereditary vitamin D–dependent rickets
- Vitamin D-resistant rickets
- Whipple disease
- X-linked familial hypophosphatemia
Causes by organ system
Causes in alphabetical order
The unnamed parameter 2= is no longer supported. Please see the documentation for {{columns-list}}.
3References
- ↑ 1.0 1.1 Hossein-nezhad A, Holick MF (2013). "Vitamin D for health: a global perspective". Mayo Clin Proc. 88 (7): 720–55. doi:10.1016/j.mayocp.2013.05.011. PMC 3761874. PMID 23790560.
- ↑ Pack AM, Morrell MJ (2004). "Epilepsy and bone health in adults". Epilepsy Behav. 5 Suppl 2: S24–9. doi:10.1016/j.yebeh.2003.11.029. PMID 15123008.
- ↑ Zhou C, Assem M, Tay JC, Watkins PB, Blumberg B, Schuetz EG; et al. (2006). "Steroid and xenobiotic receptor and vitamin D receptor crosstalk mediates CYP24 expression and drug-induced osteomalacia". J Clin Invest. 116 (6): 1703–12. doi:10.1172/JCI27793. PMC 1459072. PMID 16691293.
- ↑ Kyriakidou-Himonas M, Aloia JF, Yeh JK (1999). "Vitamin D supplementation in postmenopausal black women". J Clin Endocrinol Metab. 84 (11): 3988–90. doi:10.1210/jcem.84.11.6132. PMID 10566638.
- ↑ Holick MF (2012). "Vitamin D: extraskeletal health". Rheum Dis Clin North Am. 38 (1): 141–60. doi:10.1016/j.rdc.2012.03.013. PMID 22525849.
- ↑ Ali FN, Arguelles LM, Langman CB, Price HE (2009). "Vitamin D deficiency in children with chronic kidney disease: uncovering an epidemic". Pediatrics. 123 (3): 791–6. doi:10.1542/peds.2008-0634. PMID 19255004.
- ↑ Pappa HM, Gordon CM, Saslowsky TM, Zholudev A, Horr B, Shih MC; et al. (2006). "Vitamin D status in children and young adults with inflammatory bowel disease". Pediatrics. 118 (5): 1950–61. doi:10.1542/peds.2006-0841. PMC 3205440. PMID 17079566.
- ↑ Lo CW, Paris PW, Clemens TL, Nolan J, Holick MF (1985). "Vitamin D absorption in healthy subjects and in patients with intestinal malabsorption syndromes". Am J Clin Nutr. 42 (4): 644–9. PMID 4050723.