Hypoaldosteronism physical examination: Difference between revisions

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==Physical Examination==
==Physical Examination==
Physical examination of patients with hypoaldosteronism is usually unremarkable, unless there is severe hyperkalemia. The physical findings may represent underlying condition such as chronic kidney disease or diabetic nephropathy. Increased level of serum potassium level may present with muscle weakness and cardiac arrhythmias.
[[Patients]] with hypoaldosteronism usually appear [[Fatigue|fatigued]]. [[Physical examination]] of [[patients]] with hypoaldosteronism is usually unremarkable, unless there is severe [[hyperkalemia]]. Increased level of serum [[potassium]] level may present with muscle [[tenderness]], [[hyporeflexia]]/[[areflexia]] and [[cardiac arrhythmias]]. The physical exam may also represent findings of underlying condition such as [[chronic kidney disease]] or [[diabetic nephropathy]]


===Appearance of the Patient===
===Appearance of the Patient===

Revision as of 15:36, 31 August 2017

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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Akshun Kalia M.B.B.S.[2]

Overview

Patients with hypoaldosteronism usually appear fatigued. Physical examination of patients with hypoaldosteronism is usually unremarkable, unless there is severe hyperkalemia. Increased level of serum potassium level may present with muscle tenderness, hyporeflexia/areflexia and cardiac arrhythmias. The physical exam may also represent findings of underlying condition such as chronic kidney disease or diabetic nephropathy.

Physical Examination

Patients with hypoaldosteronism usually appear fatigued. Physical examination of patients with hypoaldosteronism is usually unremarkable, unless there is severe hyperkalemia. Increased level of serum potassium level may present with muscle tenderness, hyporeflexia/areflexia and cardiac arrhythmias. The physical exam may also represent findings of underlying condition such as chronic kidney disease or diabetic nephropathy

Appearance of the Patient

  • Patients with hypoaldosteronism usually appear fatigued.

Vital Signs

Skin

If hypoaldosteronism is from addison's disease, changes in skin and hair may be observed such as:

  • Pigmented skin and mucous membranes - darkening (hyperpigmentation) of the skin, including areas not exposed to the sun; characteristic sites are skin creases (e.g. of the hands), nipples, and the inside of the cheek (buccal mucosa), also old scars may darken.
  • Absence of axillary and pubic hair in females as a result of loss of adrenal androgens.

Heart

Hyperkalemia can lead to:

  • Irregular heart rate
  • Bradycardia
  • Heart block

Neuromuscular

  • Hyponatremia is unusual in isolated hypoaldosteronism since ADH is under inhibitory control of cortisol. However, in patients of Addison's disease there is decreased level of cortisol and aldosterone. Since there is no inhibition of ADH from cortisol, this leads to increased free water absorption and hyponatremia. Patients with hyponatremia may present with confusion when serum sodium level is <115 mmol/L
  • Hyporeflexia/areflexia

Extremities

  • Muscle weakness
  • Muscle tenderness
  • Fasciculations
  • Depressed deep tendon reflexes

References

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