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{{CMG}}; {{AE}} {{Anmol}} | {{CMG}}; {{AE}} {{Anmol}} | ||
= | =Pathogenesis= | ||
GSD type 1 results due to defects in the function of microsomal enzyme glucose-6-phosphatase (G6Pase). | |||
G6Pase catalyzes the conversion of glucose-6-phosphate to glucose during glycogenolysis and gluconeogenesis. | |||
==References== | ==References== | ||
{{reflist|2}} | {{reflist|2}} | ||
Revision as of 15:53, 30 October 2017
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Hyperparathyroidism Microchapters |
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Diagnosis |
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Treatment |
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Case Studies |
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Sandbox : anmol On the Web |
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American Roentgen Ray Society Images of Sandbox : anmol |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Anmol Pitliya, M.B.B.S. M.D.[2]
Pathogenesis
GSD type 1 results due to defects in the function of microsomal enzyme glucose-6-phosphatase (G6Pase). G6Pase catalyzes the conversion of glucose-6-phosphate to glucose during glycogenolysis and gluconeogenesis.