Oral cancer risk factors: Difference between revisions
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{{CMG}};{{AE}} {{SSW}}, {{Simrat}} | {{CMG}};{{AE}} {{SSW}}, {{Simrat}} | ||
==Overview== | ==Overview== | ||
The most potent risk factor in the development of oral cancer is [[alcohol]] intake and [[tobacco use]]. The other risk factors include male gender, age over 55 year, ultraviolet light, [[Fanconi anemia]], [[dyskeratosis congenita]], [[HPV]] infection, [[graft-versus-host disease]] (GVHD), mouthwash and irritation from dentures. | The most potent [[risk factor]] in the development of oral cancer is [[alcohol]] intake and [[tobacco use]]. The other [[risk factors]] include male gender, age over 55 year, [[ultraviolet light]], [[Fanconi anemia]], [[dyskeratosis congenita]], [[HPV]] infection, [[graft-versus-host disease]] (GVHD), mouthwash and irritation from dentures. | ||
==Risk Factors== | ==Risk Factors== | ||
* The most potent risk factor in the development of oral cavity cancer is tobacco and alcohol use. | |||
* Other risk factors include: | === Common risk factors === | ||
* The most potent [[risk factor]] in the development of oral cavity cancer is [[tobacco]] and [[Alcoholism|alcohol use]]. | |||
* Other [[risk factors]] include: | |||
**Lifestyle | **Lifestyle | ||
***Betel quid use<ref name="pmid17389167">{{cite journal |vauthors=Su CC, Yang HF, Huang SJ, Lian IeB |title=Distinctive features of oral cancer in Changhua County: high incidence, buccal mucosa preponderance, and a close relation to betel quid chewing habit |journal=J. Formos. Med. Assoc. |volume=106 |issue=3 |pages=225–33 |year=2007 |pmid=17389167 |doi= |url=}}</ref> | ***Betel quid use<ref name="pmid17389167">{{cite journal |vauthors=Su CC, Yang HF, Huang SJ, Lian IeB |title=Distinctive features of oral cancer in Changhua County: high incidence, buccal mucosa preponderance, and a close relation to betel quid chewing habit |journal=J. Formos. Med. Assoc. |volume=106 |issue=3 |pages=225–33 |year=2007 |pmid=17389167 |doi= |url=}}</ref> | ||
| Line 13: | Line 15: | ||
***[[Fanconi anemia]] | ***[[Fanconi anemia]] | ||
***[[Dyskeratosis congenita]] | ***[[Dyskeratosis congenita]] | ||
***Family history of [[squamous cell carcinoma]] | ***[[Family history]] of [[squamous cell carcinoma]] | ||
*Infective agents: | *Infective agents: | ||
**[[Human papillomavirus]] (HPV) | **[[Human papillomavirus]] ([[HPV]]) | ||
**''[[Candida albicans]]'' | **''[[Candida albicans]]'' | ||
*General | *General | ||
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**Age over 55 year | **Age over 55 year | ||
**[[Graft-versus-host disease]](GVHD) | **[[Graft-versus-host disease]](GVHD) | ||
**Immune system suppression | **[[Immunosupression|Immune system suppression]] | ||
**[[Lichen planus]] | **[[Lichen planus]] | ||
*Less common risk factors: | |||
=== Less common risk factors === | |||
*Less common [[risk factors]] for the development of oral cancer include: | |||
**According to a study: poor dental hygiene or health and use of dentures made out of metal can lead to increased risk of oral cancer.<ref name="pmid16303683">{{cite journal |vauthors=Rosenquist K, Wennerberg J, Schildt EB, Bladström A, Göran Hansson B, Andersson G |title=Oral status, oral infections and some lifestyle factors as risk factors for oral and oropharyngeal squamous cell carcinoma. A population-based case-control study in southern Sweden |journal=Acta Otolaryngol. |volume=125 |issue=12 |pages=1327–36 |year=2005 |pmid=16303683 |doi=10.1080/00016480510012273 |url=}}</ref> | **According to a study: poor dental hygiene or health and use of dentures made out of metal can lead to increased risk of oral cancer.<ref name="pmid16303683">{{cite journal |vauthors=Rosenquist K, Wennerberg J, Schildt EB, Bladström A, Göran Hansson B, Andersson G |title=Oral status, oral infections and some lifestyle factors as risk factors for oral and oropharyngeal squamous cell carcinoma. A population-based case-control study in southern Sweden |journal=Acta Otolaryngol. |volume=125 |issue=12 |pages=1327–36 |year=2005 |pmid=16303683 |doi=10.1080/00016480510012273 |url=}}</ref> | ||
**Diet | **Diet | ||
Revision as of 15:21, 22 February 2018
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1];Associate Editor(s)-in-Chief: Sargun Singh Walia M.B.B.S.[2], Simrat Sarai, M.D. [3]
Overview
The most potent risk factor in the development of oral cancer is alcohol intake and tobacco use. The other risk factors include male gender, age over 55 year, ultraviolet light, Fanconi anemia, dyskeratosis congenita, HPV infection, graft-versus-host disease (GVHD), mouthwash and irritation from dentures.
Risk Factors
Common risk factors
- The most potent risk factor in the development of oral cavity cancer is tobacco and alcohol use.
- Other risk factors include:
- Lifestyle
- Betel quid use[1]
- Genetics
- Lifestyle
- Infective agents:
- General
- Male gender
- Ultraviolet light
- Age over 55 year
- Graft-versus-host disease(GVHD)
- Immune system suppression
- Lichen planus
Less common risk factors
- Less common risk factors for the development of oral cancer include:
References
- ↑ Su CC, Yang HF, Huang SJ, Lian I (2007). "Distinctive features of oral cancer in Changhua County: high incidence, buccal mucosa preponderance, and a close relation to betel quid chewing habit". J. Formos. Med. Assoc. 106 (3): 225–33. PMID 17389167. Vancouver style error: initials (help)
- ↑ Rosenquist K, Wennerberg J, Schildt EB, Bladström A, Göran Hansson B, Andersson G (2005). "Oral status, oral infections and some lifestyle factors as risk factors for oral and oropharyngeal squamous cell carcinoma. A population-based case-control study in southern Sweden". Acta Otolaryngol. 125 (12): 1327–36. doi:10.1080/00016480510012273. PMID 16303683.
- ↑ Scully C (2002). "Oral squamous cell carcinoma; from an hypothesis about a virus, to concern about possible sexual transmission". Oral Oncol. 38 (3): 227–34. PMID 11978544.