Pulmonary edema laboratory tests: Difference between revisions
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==== ABGs: ==== | ==== ABGs: ==== | ||
Hypoxia | * [[Hypoxia]] : | ||
** [[Oxygen saturation]] < 90% | |||
** [[PaO2]] < 60 mm Hg) | |||
* [[Hypercapnia]]: | |||
** [[CO2]] > 45–55 mm Hg | |||
* [[Acidosis]]: | |||
** PH < 7.35 nEq/liter | |||
** Early findings of pulmonary edema may be [[respiratory alkalosis]] because of [[hyperventilation]] | |||
==== Serum albumin: ==== | ==== Serum albumin: ==== | ||
Revision as of 14:29, 23 February 2018
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] Associate Editor(s)-in-Chief: Farnaz Khalighinejad, MD [2]
Overview
Laboratory Findings
Laboratory findings consistent with the diagnosis of pulmonary edema include:[1][2]
ABGs:
- Hypoxia :
- Oxygen saturation < 90%
- PaO2 < 60 mm Hg)
- Hypercapnia:
- CO2 > 45–55 mm Hg
- Acidosis:
- PH < 7.35 nEq/liter
- Early findings of pulmonary edema may be respiratory alkalosis because of hyperventilation
Serum albumin:
Liver function tests:
Plasma brain natriuretic peptide levels :
B-type natriuretic peptide (BNP) is elevated in the patient with cardiogenic pulmonary edema. A low BNP (<100 pg/ml) makes a cardiac cause very unlikely and is associated with non-cardiogenic pulmonary edema.
| Laboratory findings | ECG | CXR | Cardiac enzymes | PCWP | QS/QT | Edema fluid/serum protein |
|---|---|---|---|---|---|---|
| Cardiogenic pulmonary edema | Ischemia/Infarct | Peri-hilar distribution | May be elevated | >18 mmHg | Small elevated | <.5 |
| Noncardiogenic pulmonary edema | Usually normal | Peripheral distributions | Usually normal | <18 mmHg | Large elevated | <.7 |
References
- ↑ Ware LB, Matthay MA (December 2005). "Clinical practice. Acute pulmonary edema". N. Engl. J. Med. 353 (26): 2788–96. doi:10.1056/NEJMcp052699. PMID 16382065.
- ↑ Sibbald WJ, Cunningham DR, Chin DN (October 1983). "Non-cardiac or cardiac pulmonary edema? A practical approach to clinical differentiation in critically ill patients". Chest. 84 (4): 452–61. PMID 6617283.