Multiple sclerosis other diagnostic studies: Difference between revisions
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==Other Diagnostic Studies== | ==Other Diagnostic Studies== | ||
====Visual evoked potential studies==== | ====Visual evoked potential studies==== | ||
Delay in response after stimulation of [[retina]] with light is an indicator of a problem in visual tracts due to [[Axonal|axona]]<nowiki/>l [[demyelination]]. | Delay in response after stimulation of [[retina]] with light is an indicator of a problem in visual tracts due to [[Axonal|axona]]<nowiki/>l [[demyelination]].<ref name="pmid18825673">{{cite journal |vauthors=Klistorner A, Arvind H, Nguyen T, Garrick R, Paine M, Graham S, O'Day J, Grigg J, Billson F, Yiannikas C |title=Axonal loss and myelin in early ON loss in postacute optic neuritis |journal=Ann. Neurol. |volume=64 |issue=3 |pages=325–31 |date=September 2008 |pmid=18825673 |doi=10.1002/ana.21474 |url=}}</ref> The indication of this test is when patient is not fulfilling MS criteria and is a probable MS case.<ref name="pmid3070342">{{cite journal |vauthors=Chiappa KH |title=Use of evoked potentials for diagnosis of multiple sclerosis |journal=Neurol Clin |volume=6 |issue=4 |pages=861–80 |date=November 1988 |pmid=3070342 |doi= |url=}}</ref><ref name="pmid7077339">{{cite journal |vauthors=Matthews WB, Wattam-Bell JR, Pountney E |title=Evoked potentials in the diagnosis of multiple sclerosis: a follow up study |journal=J. Neurol. Neurosurg. Psychiatry |volume=45 |issue=4 |pages=303–7 |date=April 1982 |pmid=7077339 |pmc=491364 |doi= |url=}}</ref> | ||
==== Antimyelin antibodies ==== | ==== Antimyelin antibodies ==== | ||
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Overview
Other Diagnostic Studies
Visual evoked potential studies
Delay in response after stimulation of retina with light is an indicator of a problem in visual tracts due to axonal demyelination.[1] The indication of this test is when patient is not fulfilling MS criteria and is a probable MS case.[2][3]
Antimyelin antibodies
myelin oligodendrocyte glycoprotein (MOG) and myelin basic protein (MBP) thought to be a predictor of disease progression but some studies denied any relationship between these auto antibodies and disease severity or progression.[4][5][6][7]
References
- ↑ Klistorner A, Arvind H, Nguyen T, Garrick R, Paine M, Graham S, O'Day J, Grigg J, Billson F, Yiannikas C (September 2008). "Axonal loss and myelin in early ON loss in postacute optic neuritis". Ann. Neurol. 64 (3): 325–31. doi:10.1002/ana.21474. PMID 18825673.
- ↑ Chiappa KH (November 1988). "Use of evoked potentials for diagnosis of multiple sclerosis". Neurol Clin. 6 (4): 861–80. PMID 3070342.
- ↑ Matthews WB, Wattam-Bell JR, Pountney E (April 1982). "Evoked potentials in the diagnosis of multiple sclerosis: a follow up study". J. Neurol. Neurosurg. Psychiatry. 45 (4): 303–7. PMC 491364. PMID 7077339.
- ↑ Berger T, Rubner P, Schautzer F, Egg R, Ulmer H, Mayringer I, Dilitz E, Deisenhammer F, Reindl M (July 2003). "Antimyelin antibodies as a predictor of clinically definite multiple sclerosis after a first demyelinating event". N. Engl. J. Med. 349 (2): 139–45. doi:10.1056/NEJMoa022328. PMID 12853586.
- ↑ Gaertner S, de Graaf KL, Greve B, Weissert R (December 2004). "Antibodies against glycosylated native MOG are elevated in patients with multiple sclerosis". Neurology. 63 (12): 2381–3. PMID 15623705.
- ↑ Kuhle J, Pohl C, Mehling M, Edan G, Freedman MS, Hartung HP, Polman CH, Miller DH, Montalban X, Barkhof F, Bauer L, Dahms S, Lindberg R, Kappos L, Sandbrink R (January 2007). "Lack of association between antimyelin antibodies and progression to multiple sclerosis". N. Engl. J. Med. 356 (4): 371–8. doi:10.1056/NEJMoa063602. PMID 17251533.
- ↑ Lampasona V, Franciotta D, Furlan R, Zanaboni S, Fazio R, Bonifacio E, Comi G, Martino G (June 2004). "Similar low frequency of anti-MOG IgG and IgM in MS patients and healthy subjects". Neurology. 62 (11): 2092–4. PMID 15184621.