Myasthenia gravis pathophysiology: Difference between revisions
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==Pathophysiology== | ==Pathophysiology== | ||
Myasthenia gravis is a [[neuromuscular disease]] caused by an [[autoimmune]] reactions. The main problem in this disease is the abnormal transmission of [[nerve impulses]] to [[muscle fibers]] in [[Neuromuscular junction|NMJ]]. | Myasthenia gravis is a [[neuromuscular disease]] caused by an [[autoimmune]] reactions. The main problem in this disease is the abnormal transmission of [[nerve impulses]] to [[muscle fibers]] in [[Neuromuscular junction|NMJ]].<ref name="pmid11231638">{{cite journal |vauthors=Hoch W, McConville J, Helms S, Newsom-Davis J, Melms A, Vincent A |title=Auto-antibodies to the receptor tyrosine kinase MuSK in patients with myasthenia gravis without acetylcholine receptor antibodies |journal=Nat. Med. |volume=7 |issue=3 |pages=365–8 |date=March 2001 |pmid=11231638 |doi=10.1038/85520 |url=}}</ref> | ||
==References== | ==References== | ||
{{Reflist|2}} | {{Reflist|2}} |
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Overview
Pathophysiology
Myasthenia gravis is a neuromuscular disease caused by an autoimmune reactions. The main problem in this disease is the abnormal transmission of nerve impulses to muscle fibers in NMJ.[1]
References
- ↑ Hoch W, McConville J, Helms S, Newsom-Davis J, Melms A, Vincent A (March 2001). "Auto-antibodies to the receptor tyrosine kinase MuSK in patients with myasthenia gravis without acetylcholine receptor antibodies". Nat. Med. 7 (3): 365–8. doi:10.1038/85520. PMID 11231638.