Scrotal mass pathophysiology: Difference between revisions
No edit summary |
|||
| Line 12: | Line 12: | ||
===Phatogenesis=== | ===Phatogenesis=== | ||
*It is understood that varicocele is the result of dilation of pampiniform venous plexus along spermatic cord. | |||
*The pathophysiology of testicular cancer depends on the histological subtype. | |||
*Testicular tortion is produced by Inadequate [[Fixation (visual)|fixation]] of the [[Lower gastrointestinal bleeding resident survival guide|lower pole]] of the [[testis]] to the [[tunica vaginalis]] causes testicular tortion . If [[fixation]] is absent , the testis may torse (twist) on the [[Spermatic cord|spermatic cord,]] lead to produce[[ischemia]] from reduced [[Arterial insufficiency|arterial inflow]] and [[venous]] [[Outflow tract VT|outflow]] [[obstruction]] | |||
==Genetics== | ==Genetics== | ||
Revision as of 16:13, 31 October 2019
|
Scrotal Mass Microchapters |
|
Diagnosis |
|---|
|
Treatment |
|
Case Studies |
|
Scrotal mass pathophysiology On the Web |
|
American Roentgen Ray Society Images of Scrotal mass pathophysiology |
|
Risk calculators and risk factors for Scrotal mass pathophysiology |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]Associate Editor(s)-in-Chief: Niloofarsadaat Eshaghhosseiny, MD[2]
Overview
Deponds on the causes phatophysiology is different.Inadequate fixation of the lower pole of the testis to the tunica vaginalis causes testicular tortion . If fixation is absent , the testis may torse (twist) on the spermatic cord, lead to produceischemia from reduced arterial inflow and venous outflow obstruction . Testicular torsion etiology include (eg, trauma, vigorous physical activity) or spontaneously. Acquired hernias due loss of mechanical integrity of the abdominal wall muscles and tendons . primary hernia due Genetic or systemic extracellular matrix disorders and defective wound healing after laparotomy and hernia repairs may predispose to incisional hernias.
Pathophisiology
Physiology
Scrotal is extention of abdominal wall contains testis , tunica vaginalis ,spermatic cord,epididimysis and appendix testis.Testis has seminiferous tubules that has germ cells,sertoli cells, and leydig cells .Germ cells develop into spermatogenesis and produce gametes.Sertoli secrete inhibin and leydig cell produce testostrone.[1]The normal physiology of germ cells is production of gametes which are reproductive cells.After migration of these cells to gonads, they udergo meiosis to produce gametes.[2]
Phatogenesis
- It is understood that varicocele is the result of dilation of pampiniform venous plexus along spermatic cord.
- The pathophysiology of testicular cancer depends on the histological subtype.
- Testicular tortion is produced by Inadequate fixation of the lower pole of the testis to the tunica vaginalis causes testicular tortion . If fixation is absent , the testis may torse (twist) on the spermatic cord, lead to produceischemia from reduced arterial inflow and venous outflow obstruction
Genetics
Associated Conditions
Gros Pathology
Microscopic Pathology
References
- ↑ Djureinovic D, Fagerberg L, Hallström B, Danielsson A, Lindskog C, Uhlén M; et al. (2014). "The human testis-specific proteome defined by transcriptomics and antibody-based profiling". Mol Hum Reprod. 20 (6): 476–88. doi:10.1093/molehr/gau018. PMID 24598113.
- ↑ Cinalli RM, Rangan P, Lehmann R (2008). "Germ cells are forever". Cell. 132 (4): 559–62. doi:10.1016/j.cell.2008.02.003. PMID 18295574.