Endocardial cushion defect pathophysiology: Difference between revisions
Aditya Ganti (talk | contribs) Created page with "__NOTOC__ {{Endocardial cushion defect}} {{CMG}}; {{AE}} {{ADG}} ==Overview== The exact pathogenesis of [disease name] is not fully understood. OR It is thought that [disea..." |
Aditya Ganti (talk | contribs) |
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==Associated Conditions== | ==Associated Conditions== | ||
Conditions associated with | Conditions associated with endocardial cushion defect include: | ||
*[ | *[[Tetralogy of Fallot]] | ||
*[ | *[[Transposition of the great vessels|Transposition of the great arteries]] | ||
*[ | *[[Patent ductus arteriosus]] | ||
*[[Coarctation of the aorta]] | |||
*Absent atrial septum | |||
*[[Persistent left superior vena cava]] | |||
*[[Anomalous pulmonary venous connection]] | |||
==Gross Pathology== | ==Gross Pathology== |
Revision as of 07:58, 20 April 2020
Endocardial cushion defect Microchapters |
Differentiating Endocardial cushion defect from other Diseases |
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Endocardial cushion defect pathophysiology On the Web |
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Risk calculators and risk factors for Endocardial cushion defect pathophysiology |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Aditya Ganti M.B.B.S. [2]
Overview
The exact pathogenesis of [disease name] is not fully understood.
OR
It is thought that [disease name] is the result of / is mediated by / is produced by / is caused by either [hypothesis 1], [hypothesis 2], or [hypothesis 3].
OR
[Pathogen name] is usually transmitted via the [transmission route] route to the human host.
OR
Following transmission/ingestion, the [pathogen] uses the [entry site] to invade the [cell name] cell.
OR
[Disease or malignancy name] arises from [cell name]s, which are [cell type] cells that are normally involved in [function of cells].
OR
The progression to [disease name] usually involves the [molecular pathway].
OR
The pathophysiology of [disease/malignancy] depends on the histological subtype.
Pathophysiology
Physiology
The normal physiology of [name of process] can be understood as follows:
Pathogenesis
- The exact pathogenesis of [disease name] is not completely understood.
OR
- It is understood that [disease name] is the result of / is mediated by / is produced by / is caused by either [hypothesis 1], [hypothesis 2], or [hypothesis 3].
- [Pathogen name] is usually transmitted via the [transmission route] route to the human host.
- Following transmission/ingestion, the [pathogen] uses the [entry site] to invade the [cell name] cell.
- [Disease or malignancy name] arises from [cell name]s, which are [cell type] cells that are normally involved in [function of cells].
- The progression to [disease name] usually involves the [molecular pathway].
- The pathophysiology of [disease/malignancy] depends on the histological subtype.
Genetics
[Disease name] is transmitted in [mode of genetic transmission] pattern.
OR
Genes involved in the pathogenesis of [disease name] include:
- [Gene1]
- [Gene2]
- [Gene3]
OR
The development of [disease name] is the result of multiple genetic mutations such as:
- [Mutation 1]
- [Mutation 2]
- [Mutation 3]
Associated Conditions
Conditions associated with endocardial cushion defect include:
- Tetralogy of Fallot
- Transposition of the great arteries
- Patent ductus arteriosus
- Coarctation of the aorta
- Absent atrial septum
- Persistent left superior vena cava
- Anomalous pulmonary venous connection
Gross Pathology
On gross pathology, [feature1], [feature2], and [feature3] are characteristic findings of [disease name].
Microscopic Pathology
On microscopic histopathological analysis, [feature1], [feature2], and [feature3] are characteristic findings of [disease name].