Non-bacterial thrombotic endocarditis risk factors: Difference between revisions

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==Risk Factors==
==Risk Factors==
The most potent risk factor in the development of non-bacterial thrombotic endocarditis is advanced malignancy<ref name="pmid4682494">{{cite journal |vauthors=Rosen P, Armstrong D |title=Nonbacterial thrombotic endocarditis in patients with malignant neoplastic diseases |journal=Am. J. Med. |volume=54 |issue=1 |pages=23–9 |date=January 1973 |pmid=4682494 |doi=10.1016/0002-9343(73)90079-x |url=}}</ref><ref name="pmid4682494">{{cite journal |vauthors=Rosen P, Armstrong D |title=Nonbacterial thrombotic endocarditis in patients with malignant neoplastic diseases |journal=Am. J. Med. |volume=54 |issue=1 |pages=23–9 |date=January 1973 |pmid=4682494 |doi=10.1016/0002-9343(73)90079-x |url=}}</ref>. Other risk factors include systemic lupus erythematosus, antiphospholipid syndrome <ref>Roldan CA, Qualls CR, Sopko KS, Sibbitt WL Jr. Transthoracic versus transesophageal echocardiography for detection of Libman-Sacks endocarditis: A randomized controlled study. The Journal of Rheumatology. 2008;35:224</ref>, and chronic inflammatory states.
The most potent risk factor in the development of non-bacterial thrombotic endocarditis is advanced malignancy<ref name="pmid1851590">{{cite journal |vauthors=González Quintela A, Candela MJ, Vidal C, Román J, Aramburo P |title=Non-bacterial thrombotic endocarditis in cancer patients |journal=Acta Cardiol |volume=46 |issue=1 |pages=1–9 |date=1991 |pmid=1851590 |doi= |url=}}</ref><ref name="pmid4682494">{{cite journal |vauthors=Rosen P, Armstrong D |title=Nonbacterial thrombotic endocarditis in patients with malignant neoplastic diseases |journal=Am. J. Med. |volume=54 |issue=1 |pages=23–9 |date=January 1973 |pmid=4682494 |doi=10.1016/0002-9343(73)90079-x |url=}}</ref>. Other risk factors include systemic lupus erythematosus, antiphospholipid syndrome <ref>Roldan CA, Qualls CR, Sopko KS, Sibbitt WL Jr. Transthoracic versus transesophageal echocardiography for detection of Libman-Sacks endocarditis: A randomized controlled study. The Journal of Rheumatology. 2008;35:224</ref>, and chronic inflammatory states.


==References==
==References==

Revision as of 06:31, 7 August 2020

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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Aisha Adigun, B.Sc., M.D.[2]

Overview

The most potent risk factor in the development of non-bacterial thrombotic endocarditis is advanced malignancy. Other risk factors include systemic lupus erythematosus, antiphospholipid syndrome, and chronic inflammatory states.

Risk Factors

The most potent risk factor in the development of non-bacterial thrombotic endocarditis is advanced malignancy[1][2]. Other risk factors include systemic lupus erythematosus, antiphospholipid syndrome [3], and chronic inflammatory states.

References

  1. González Quintela A, Candela MJ, Vidal C, Román J, Aramburo P (1991). "Non-bacterial thrombotic endocarditis in cancer patients". Acta Cardiol. 46 (1): 1–9. PMID 1851590.
  2. Rosen P, Armstrong D (January 1973). "Nonbacterial thrombotic endocarditis in patients with malignant neoplastic diseases". Am. J. Med. 54 (1): 23–9. doi:10.1016/0002-9343(73)90079-x. PMID 4682494.
  3. Roldan CA, Qualls CR, Sopko KS, Sibbitt WL Jr. Transthoracic versus transesophageal echocardiography for detection of Libman-Sacks endocarditis: A randomized controlled study. The Journal of Rheumatology. 2008;35:224

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