Tricuspid stenosis causes: Difference between revisions
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== | ==Causes of Tricuspid Stenosis== | ||
There are at least 4 conditions that are typically attributed with the obstruction of the native[[ tricuspid valve]]. | |||
# Rheumatic tricuspid stenosis: | |||
#* Diffuse thickening of the leaflets occur. Fusion of the commissures may or may not occur. | |||
#* Chordae tendineae may become thickened and shortened | |||
#* As a result of the dense collagen and elastic fibers that make up leaflet tissue, the normal leaflet layers become significantly distorted | |||
# Carcinoid heart disease: | |||
#* Fibrous white plaques located on the valvular and mural endocardium are characteristic presentations of carcinoid valve lesions | |||
#* Valve leaflets become thick, rigid and smaller in area | |||
#* Atrial and ventricular surfaces of the valve structure contain fibrous tissue proliferation | |||
# Congenital tricuspid stenosis: | |||
#* More common in infants | |||
#* Lesions may present in a number of different ways, either singularly or in any combination of the following: | |||
#** Incompletely developed leaflets | |||
#** Shortened or malformed chordae | |||
#** Small annuli | |||
#** Papillary muscles of abnormal size and number | |||
# Infective endocarditis: | |||
#* Stenosis may develop as a result of large infected vegetation obstructing the opening of the tricuspid valve | |||
#* This condition is uncommon | |||
* Unusual and rare causes: | |||
** Fabry disease | |||
** Giant blood cysts | |||
* Other conditions may mimic tricuspid stenosis, obstructing flow through the valve: | |||
** Supravalvular obstruction from congenital diaphragms | |||
** Intracardiac or extracardiac tumors | |||
** Thrombosis or emboli | |||
** Large endocarditis vegetations | |||
** Other conditions that impair right-sided filling | |||
*** Constrictive pericarditis | |||
*** Restrictive Cardiomyopathy | |||
==References== | ==References== | ||
Revision as of 19:43, 19 July 2011
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Causes of Tricuspid Stenosis
There are at least 4 conditions that are typically attributed with the obstruction of the nativetricuspid valve.
- Rheumatic tricuspid stenosis:
- Diffuse thickening of the leaflets occur. Fusion of the commissures may or may not occur.
- Chordae tendineae may become thickened and shortened
- As a result of the dense collagen and elastic fibers that make up leaflet tissue, the normal leaflet layers become significantly distorted
- Carcinoid heart disease:
- Fibrous white plaques located on the valvular and mural endocardium are characteristic presentations of carcinoid valve lesions
- Valve leaflets become thick, rigid and smaller in area
- Atrial and ventricular surfaces of the valve structure contain fibrous tissue proliferation
- Congenital tricuspid stenosis:
- More common in infants
- Lesions may present in a number of different ways, either singularly or in any combination of the following:
- Incompletely developed leaflets
- Shortened or malformed chordae
- Small annuli
- Papillary muscles of abnormal size and number
- Infective endocarditis:
- Stenosis may develop as a result of large infected vegetation obstructing the opening of the tricuspid valve
- This condition is uncommon
- Unusual and rare causes:
- Fabry disease
- Giant blood cysts
- Other conditions may mimic tricuspid stenosis, obstructing flow through the valve:
- Supravalvular obstruction from congenital diaphragms
- Intracardiac or extracardiac tumors
- Thrombosis or emboli
- Large endocarditis vegetations
- Other conditions that impair right-sided filling
- Constrictive pericarditis
- Restrictive Cardiomyopathy