Papilledema pathophysiology: Difference between revisions
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{{CMG}}''' Associate Editor(s)-In-Chief''':Kalsang Dolma | {{CMG}}''' Associate Editor(s)-In-Chief''':Kalsang Dolma | ||
==Overview== | ==Overview== | ||
[[Papilledema]] results due to transmission of [[increased intracranial pressure]] to the anterior end of [[optic nerve]] through optic nerve sheath. | |||
==Pathophysiology== | ==Pathophysiology== | ||
Revision as of 18:25, 17 July 2012
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Papilledema |
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Diagnosis |
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Treatment |
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Papilledema pathophysiology On the Web |
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American Roentgen Ray Society Images of Papilledema pathophysiology |
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Risk calculators and risk factors for Papilledema pathophysiology |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] Associate Editor(s)-In-Chief:Kalsang Dolma
Overview
Papilledema results due to transmission of increased intracranial pressure to the anterior end of optic nerve through optic nerve sheath.
Pathophysiology
- Optic nerve sheath is continuous with the subarachnoid space of the brain and is regarded as an extension of the central nervous system.
- The brain itself is relatively spared from pathological consequences of high pressure.
- Increased intracranial pressure is transmitted through to the optic nerve via this optic nerve sheath.
- The anterior end of the optic nerve stops abruptly at the eye. Hence the pressure is asymmetrical and this causes a pinching and protrusion of the optic nerve at its head.
- The fibers of the retinal ganglion cells of the optic disc become engorged and bulge anteriorly.
- Persistent and extensive optic nerve head swelling, or optic disc edema, can lead to loss of these fibers and permanent visual impairment.