Miosis pathophysiology: Difference between revisions
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== | ==Pathophysiology== | ||
Visual stimuli enters the eye,and through the retinal photoceptors is converted into an electric impulse, carried through the optic nerve ([[cranial nerve]] II) to the brain, where it connects to the pretectal nucleus of the high midbrain. It bypasses the lateral geniculate nucleus and the primary visual cortex. From the pretectal nucleus neurons send axons to neurons of the [[Edinger-Westphal nucleus]] whose visceromotor axons run along both the left and right oculomotor nerves. Visceromotor nerve axons (which constitute a portion of the III [[cranial nerve]], along with the somatomotor portion derived from the Edinger-Westphal nucleus) synapse on ciliary ganglion neurons, whose [[parasympathetic]] axons innervate the constrictor muscle of the iris, producing '''[[miosis]]'''. | Visual stimuli enters the eye,and through the retinal photoceptors is converted into an electric impulse, carried through the optic nerve ([[cranial nerve]] II) to the brain, where it connects to the pretectal nucleus of the high midbrain. It bypasses the lateral geniculate nucleus and the primary visual cortex. From the pretectal nucleus neurons send axons to neurons of the [[Edinger-Westphal nucleus]] whose visceromotor axons run along both the left and right oculomotor nerves. Visceromotor nerve axons (which constitute a portion of the III [[cranial nerve]], along with the somatomotor portion derived from the Edinger-Westphal nucleus) synapse on ciliary ganglion neurons, whose [[parasympathetic]] axons innervate the constrictor muscle of the iris, producing '''[[miosis]]'''. | ||
[http://www.coldbacon.com/mdtruth/pics/netter/fn-oculomotor.jpg] | [http://www.coldbacon.com/mdtruth/pics/netter/fn-oculomotor.jpg] | ||
==References== | ==References== | ||
{{reflist|2}} | {{reflist|2}} | ||
[[Category:Signs and symptoms]] | |||
[[Category:Physical examination]] | |||
[[Category:Ophthalmology]] | |||
[[Category:Neurology]] | |||
[[Category:Emergency medicine]] | |||
[[Category:Disease]] | |||
{{WH}} | {{WH}} | ||
{{WS}} | {{WS}} | ||
Revision as of 20:32, 20 February 2013
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Miosis Microchapters |
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Miosis pathophysiology On the Web |
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Risk calculators and risk factors for Miosis pathophysiology |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Pathophysiology
Visual stimuli enters the eye,and through the retinal photoceptors is converted into an electric impulse, carried through the optic nerve (cranial nerve II) to the brain, where it connects to the pretectal nucleus of the high midbrain. It bypasses the lateral geniculate nucleus and the primary visual cortex. From the pretectal nucleus neurons send axons to neurons of the Edinger-Westphal nucleus whose visceromotor axons run along both the left and right oculomotor nerves. Visceromotor nerve axons (which constitute a portion of the III cranial nerve, along with the somatomotor portion derived from the Edinger-Westphal nucleus) synapse on ciliary ganglion neurons, whose parasympathetic axons innervate the constrictor muscle of the iris, producing miosis. [2]
![[2]](http://www.coldbacon.com/mdtruth/pics/netter/fn-oculomotor.jpg){kind=link}