Adiposogenital dystrophy history and symptoms: Difference between revisions
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==Overview== | ==Overview== | ||
==History and Symptoms== | |||
It is characterized by: | |||
* Feminine [[obesity]] | |||
* [[Growth retardation]] and retarded [[sexual development]], [[atrophy]] or [[hypoplasia]] of the [[gonads]], and altered [[secondary sex characteristics]], | |||
* [[headaches]] | |||
* [[mental retardation]], problems with [[Visual perception|vision]] | |||
* [[polyuria]], [[polydipsia]]. | |||
It is usually associated with [[tumor|tumours]] of the [[hypothalamus]], causing increased [[appetite]] and depressed [[secretion]] of [[gonadotropin]]. It seems to affect [[males]] mostly. | |||
Many overweight children may appear to have the disorder because of the concurrence of obesity and retarded sexual development; these children have no [[endocrine]] disturbances, however, and they mature normally after delayed [[puberty]]. | |||
==References== | ==References== | ||
{{reflist|2}} | {{reflist|2}} | ||
Revision as of 14:41, 20 September 2012
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Adiposogenital dystrophy Microchapters |
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Differentiating Adiposogenital dystrophy from other Diseases |
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Diagnosis |
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Treatment |
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Case Studies |
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Adiposogenital dystrophy history and symptoms On the Web |
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American Roentgen Ray Society Images of Adiposogenital dystrophy history and symptoms |
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Risk calculators and risk factors for Adiposogenital dystrophy history and symptoms |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Overview
History and Symptoms
It is characterized by:
- Feminine obesity
- Growth retardation and retarded sexual development, atrophy or hypoplasia of the gonads, and altered secondary sex characteristics,
- headaches
- mental retardation, problems with vision
- polyuria, polydipsia.
It is usually associated with tumours of the hypothalamus, causing increased appetite and depressed secretion of gonadotropin. It seems to affect males mostly.
Many overweight children may appear to have the disorder because of the concurrence of obesity and retarded sexual development; these children have no endocrine disturbances, however, and they mature normally after delayed puberty.