Hepatorenal syndrome differential diagnosis: Difference between revisions
Jump to navigation
Jump to search
No edit summary |
Irfan Dotani (talk | contribs) No edit summary |
||
Line 1: | Line 1: | ||
__NOTOC__ | __NOTOC__ | ||
{{Hepatorenal syndrome}} | {{Hepatorenal syndrome}} | ||
{{CMG}}; {{AE}} | |||
{{PleaseHelp}} | |||
{{ | |||
==Overview== | ==Overview== | ||
Line 16: | Line 15: | ||
==References== | ==References== | ||
{{Reflist|2}} | {{Reflist|2}} | ||
[[Category: | [[Category:Hepatology]] | ||
[[Category:Gastroenterology]] | [[Category:Gastroenterology]] | ||
[[Category:Nephrology]] | [[Category:Nephrology]] |
Revision as of 12:39, 18 July 2016
Hepatorenal syndrome Microchapters |
Diagnosis |
---|
Treatment |
Case Studies |
Hepatorenal syndrome differential diagnosis On the Web |
American Roentgen Ray Society Images of Hepatorenal syndrome differential diagnosis |
Risk calculators and risk factors for Hepatorenal syndrome differential diagnosis |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief:
Please help WikiDoc by adding content here. It's easy! Click here to learn about editing.
Overview
Differentiating Hepatorenal Syndrome from other Diseases
Many other diseases of the kidney are associated with liver disease and must be excluded before making a diagnosis of hepatorenal syndrome. They include the following:
- Pre-renal failure: Pre-renal failure usually responds to treatment with intravenous fluids, resulting in reduction in serum creatinine and the excretion of sodium.
- Acute tubular necrosis (ATN): This can be difficult to confidently diagnose. It may be an inability to concentrate the urine, if any is being produced. The urine sediment should be bland, microscopy may show hyaline casts. ATN may recover with supportive treatment only or progress to end-stage renal failure. In cirrhosis, urinary sodium is not a reliable guide to the development of ATN, as fractional sodium excretion may stay below 1 percent, due to the gradual worsening of renal ischaemia.
- Other causes may include glomerular disease secondary to Hepatitis B or Hepatitis C,[1] drug toxicity (notably gentamicin) or contrast nephropathy.
References
- ↑ Han SH. Extrahepatic manifestations of chronic hepatitis B. Clin Liver Dis. 2004 May;8(2):403-18. PMID 15481347