Acrodermatitis chronica atrophicans pathophysiology: Difference between revisions
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===Microscopic Pathology=== | ===Microscopic Pathology=== | ||
* Light and electron microscopic study of the [[skin biopsy]] shows degeneration of the elastica and collagen fibers.<ref name="pmid7751475">{{cite journal |author=de Koning J, Tazelaar DJ, Hoogkamp-Korstanje JA, Elema JD |title=Acrodermatitis chronica atrophicans: a light and electron microscopic study |journal=J. Cutan. Pathol. |volume=22 |issue=1 |pages=23–32 |year=1995 |month=February |pmid=7751475 |doi= |url=}}</ref> | * Light and electron microscopic study of the [[skin biopsy]] shows degeneration of the [[elastica]] and [[collagen]] fibers.<ref name="pmid7751475">{{cite journal |author=de Koning J, Tazelaar DJ, Hoogkamp-Korstanje JA, Elema JD |title=Acrodermatitis chronica atrophicans: a light and electron microscopic study |journal=J. Cutan. Pathol. |volume=22 |issue=1 |pages=23–32 |year=1995 |month=February |pmid=7751475 |doi= |url=}}</ref> | ||
==References== | ==References== | ||
Revision as of 14:03, 31 October 2012
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Raviteja Guddeti, M.B.B.S. [2]
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Pathophysiology
This progressive skin process is due to the effect of continuing active infection with the spirochete Borrelia afzelii. B afzelii is the predominant pathophysiology, but may not be the exclusive, etiologic agent of ACA. Borrelia garinii, has also been detected.
Microscopic Pathology
- Light and electron microscopic study of the skin biopsy shows degeneration of the elastica and collagen fibers.[1]