Congenital syphilis pathophysiology: Difference between revisions
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==Pathophysiology== | ==Pathophysiology== | ||
===Transmission=== | ===Transmission=== | ||
It being a congenital condition spreads through placenta from mother to fetus during pregnancy. Transmission of the disease to the fetus is largely dependent on the duration of the disease in the mother. The longer the mother has had the disease at the time of pregnancy, the chance of fetal infection decreases (40% in early latency and 10% in late latency). | It being a congenital condition spreads through placenta from mother to fetus during pregnancy. Transmission of the disease to the fetus is largely dependent on the duration of the disease in the mother. The longer the mother has had the disease at the time of pregnancy, the chance of fetal infection decreases (40% in early latency and 10% in late latency).<ref name="urlEvaluation of Molecular Methodologies and Rabbit Infectivity Testing for the Diagnosis of Congenital Syphilis and Neonatal Central Nervous System Invasion by Treponema pallidum">{{cite web |url=http://jid.oxfordjournals.org/content/167/1/148.abstract?ijkey=b63027ef9b8fdd3bfacfd69547b3f20c245266a5&keytype2=tf_ipsecsha |title=Evaluation of Molecular Methodologies and Rabbit Infectivity Testing for the Diagnosis of Congenital Syphilis and Neonatal Central Nervous System Invasion by Treponema pallidum |format= |work= |accessdate=2012-12-20}}</ref> | ||
==References== | ==References== | ||
Revision as of 20:44, 20 December 2012
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Aditya Govindavarjhulla, M.B.B.S. [2]
Overview
Pathophysiology of congenital syphilis is still unclear. Several theories have been postulated in regards to duration of infection in mother and stage of pregnancy.
Pathophysiology
Transmission
It being a congenital condition spreads through placenta from mother to fetus during pregnancy. Transmission of the disease to the fetus is largely dependent on the duration of the disease in the mother. The longer the mother has had the disease at the time of pregnancy, the chance of fetal infection decreases (40% in early latency and 10% in late latency).[1]