Premature atrial contraction laboratory findings: Difference between revisions
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==Overview== | ==Overview== | ||
Many cases of premature atrial contraction have no definite cause, it may be the result of various other problems. If [[PAC]] patients present with symptoms, a generalized approach is done to find the precipitating factors. | Many cases of premature atrial contraction have no definite cause, it may be the result of various other problems. If [[PAC]] patients present with symptoms, a generalized approach is done to find the precipitating factors. | ||
==Laboratory Findings== | ==Laboratory Findings== | ||
* [[Renal function]] and [[electrolyte]]s | The following investigations are done to patients with symptoms : | ||
* | * [[Renal function]] and [[electrolyte]]s | ||
* [[Thyroid-stimulating hormone]](commonly suppressed in [[hyperthyroidism]] and of relevance if [[amiodarone]] is administered for treatment) | * [[Complete blood count]] | ||
* [[Thyroid-stimulating hormone]](commonly suppressed in [[hyperthyroidism]] and of relevance if [[amiodarone]] is administered for treatment) | |||
==References== | ==References== |
Revision as of 22:32, 12 August 2013
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Mugilan Poongkunran M.B.B.S [2]
Overview
Many cases of premature atrial contraction have no definite cause, it may be the result of various other problems. If PAC patients present with symptoms, a generalized approach is done to find the precipitating factors.
Laboratory Findings
The following investigations are done to patients with symptoms :
- Renal function and electrolytes
- Complete blood count
- Thyroid-stimulating hormone(commonly suppressed in hyperthyroidism and of relevance if amiodarone is administered for treatment)
References