Premature atrial contraction laboratory findings: Difference between revisions
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* [[Renal function]] and [[electrolyte]]s | * [[Renal function]] and [[electrolyte]]s | ||
* [[Complete blood count]] | * [[Complete blood count]] | ||
* [[Thyroid-stimulating hormone]](commonly suppressed in [[hyperthyroidism]] and of relevance if [[amiodarone]] is administered for treatment) | * [[Thyroid-stimulating hormone]] (commonly suppressed in [[hyperthyroidism]] and of relevance if [[amiodarone]] is administered for treatment) | ||
==References== | ==References== | ||
Revision as of 22:34, 12 August 2013
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Premature atrial contraction Microchapters |
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Differentiating Premature atrial contraction from other Diseases |
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Diagnosis |
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Treatment |
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Case Studies |
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Premature atrial contraction laboratory findings On the Web |
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American Roentgen Ray Society Images of Premature atrial contraction laboratory findings |
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Risk calculators and risk factors for Premature atrial contraction laboratory findings |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Mugilan Poongkunran M.B.B.S [2]
Overview
Many cases of premature atrial contraction have no definite cause, it may be the result of various other problems. If PAC patients present with symptoms, a generalized approach is done to find the precipitating factors.
Laboratory Findings
The following investigations are done to patients with symptoms :
- Renal function and electrolytes
- Complete blood count
- Thyroid-stimulating hormone (commonly suppressed in hyperthyroidism and of relevance if amiodarone is administered for treatment)
References