Gliomatosis cerebri other imaging findings: Difference between revisions
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===4. Bone Scan=== | ===4. Bone Scan=== | ||
[[Bone scan]] may be performed to detect metastases of gliomatosis cerebri to [[bones]]. | *Most malignant gliomas get metastasized to the bones via the hematogenous spread. | ||
<ref name="pmid24212625">{{cite journal| author=Beauchesne P| title=Extra-neural metastases of malignant gliomas: myth or reality? | journal=Cancers (Basel) | year= 2011 | volume= 3 | issue= 1 | pages= 461-77 | pmid=24212625 | doi=10.3390/cancers3010461 | pmc=PMC3756372 | url=http://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=24212625 }} </ref> | |||
*[[Bone scan]] may be performed to detect metastases of gliomatosis cerebri to [[bones]]. | |||
==References== | ==References== | ||
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]Associate Editor(s)-in-Chief: Sujit Routray, M.D. [2]
Overview
Other imaging studies for gliomatosis cerebri include MR spectroscopy (decreased NAA/creatine ratio and elevated choline/creatine ratio, choline/NAA ratio, and myoinositol), MR perfusion (low/normal relative cerebral blood flow), PET scan (markedly decreased accumulation of [18F]-fluorodeoxyglucose on F-18 FDG PET, hypermetabolism on C-11 methionine PET, and marked increase in cerebral blood flow on 15(O)-water PET), and bone scan (metastasis to bones).[1][2][3]
Other Imaging Findings
Other imaging studies for gliomatosis cerebri include MR spectroscopy (decreased NAA/creatine ratio and elevated choline/creatine ratio, choline/NAA ratio, and myoinositol), MR perfusion (low/normal relative cerebral blood flow), PET scan (markedly decreased accumulation of [18F]-fluorodeoxyglucose on F-18 FDG PET, hypermetabolism on C-11 methionine PET, and marked increase in cerebral blood flow on 15(O)-water PET), and bone scan (metastasis to bones).[1][2][3]
1. MR Spectroscopy
Other imaging studies for gliomatosis cerebri include MR spectroscopy. Common findings are described below:[1][3]
- Decreased NAA/creatine ratio
- Elevated choline/creatine ratio
- Elevated choline/NAA ratio
- Elevated myoinositol
2. MR Perfusion
Other imaging studies for gliomatosis cerebri include MR perfusion. Common findings are described below:[1]
- Low/normal relative cerebral blood flow (no vascular hyperplasia)
3. PET
Other imaging studies for gliomatosis cerebri include PET scan. Common features are described below:[1][2]
- F-18 FDG PET images show markedly decreased accumulation of [18F]-fluorodeoxyglucose (glucose hypometabolism).
- C-11 methionine PET images show hypermetabolism.
- 15(O)-water PET images show a marked increase in cerebral blood flow in gliomatosis cerebri lesions.
4. Bone Scan
- Most malignant gliomas get metastasized to the bones via the hematogenous spread.
References
- ↑ 1.0 1.1 1.2 1.3 1.4 Radiographic features of gliomatosis cerebri. Dr Henry Knipe and Dr Frank Gaillard et al. Radiopaedia 2015. http://radiopaedia.org/articles/gliomatosis-cerebri
- ↑ 2.0 2.1 2.2 Rajz GG, Nass D, Talianski E, Pfeffer R, Spiegelmann R, Cohen ZR (2012). "Presentation patterns and outcome of gliomatosis cerebri". Oncol Lett. 3 (1): 209–213. doi:10.3892/ol.2011.445. PMC 3362440. PMID 22740882.
- ↑ 3.0 3.1 3.2 Desclée P, Rommel D, Hernalsteen D, Godfraind C, de Coene B, Cosnard G (2010). "Gliomatosis cerebri, imaging findings of 12 cases". J Neuroradiol. 37 (3): 148–58. doi:10.1016/j.neurad.2009.12.001. PMID 20334921.
- ↑ Beauchesne P (2011). "Extra-neural metastases of malignant gliomas: myth or reality?". Cancers (Basel). 3 (1): 461–77. doi:10.3390/cancers3010461. PMC 3756372. PMID 24212625.