Gliomatosis cerebri physical examination: Difference between revisions
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**[[Corticospinal tract|Corticospinal tract defects]] | **[[Corticospinal tract|Corticospinal tract defects]] | ||
**[[Spinocerebellar tract|Spinocerebellar tract defects]] | **[[Spinocerebellar tract|Spinocerebellar tract defects]] | ||
**Facial [[paresthesia]] suggestive of cranial nerve VII deficit | **Facial [[paresthesia]] suggestive of [[Facial nerve|cranial nerve VII deficit]] | ||
*[[Ataxia]] | *[[Ataxia]] | ||
*[[Hemiparesis]] | *[[Hemiparesis]] | ||
Revision as of 17:45, 8 October 2015
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]Associate Editor(s)-in-Chief: Sujit Routray, M.D. [2]
Overview
Common physical examination findings of gliomatosis cerebri include dysphagia, dysarthria, nystagmus, papilledema, hemiparesis, facial paresthesia, vision loss, ataxia, altered mental status, aphasia, and focal neurological defects (corticospinal tract defects, spinocerebellar tract defects, and cranioneuropathies).[1][2]
Physical Examination
Common physical examination findings of gliomatosis cerebri include:[1][2]
HEENT
- Nystagmus
- Ophthalmoscopic exam may be abnormal with findings of papilledema
- Facial paresthesia
- Vision loss
Neurological
- Patient is usually oriented to persons, place, and time
- Altered mental status
- Focal neurological defects
- Corticospinal tract defects
- Spinocerebellar tract defects
- Facial paresthesia suggestive of cranial nerve VII deficit
- Ataxia
- Hemiparesis
- Aphasia
- Dysarthria
References
- ↑ 1.0 1.1 Buis DR, van der Valk P, De Witt Hamer PC (2012). "Subcutaneous tumor seeding after biopsy in gliomatosis cerebri". J Neurooncol. 106 (2): 431–5. doi:10.1007/s11060-011-0678-2. PMC 3230756. PMID 21837541.
- ↑ 2.0 2.1 Rajz GG, Nass D, Talianski E, Pfeffer R, Spiegelmann R, Cohen ZR (2012). "Presentation patterns and outcome of gliomatosis cerebri". Oncol Lett. 3 (1): 209–213. doi:10.3892/ol.2011.445. PMC 3362440. PMID 22740882.