Congenital rubella syndrome pathophysiology: Difference between revisions

Congenital infections Main Page
	Congenital Rubella Syndrome Microchapters
Home
Patient Information
Overview
Historical Perspective
Classification
Pathophysiology
Causes
Differentiating Congenital Rubella Syndrome from other Diseases
Epidemiology and Demographics
Risk Factors
Screening
Natural History, Complications and Prognosis
Diagnosis
CDC Case Definitions
History and Symptoms
Physical Examination
Laboratory Findings
Electrocardiogram
Chest X Ray
CT
MRI
Echocardiography or Ultrasound
Other Imaging Findings
Other Diagnostic Studies
Treatment
Medical Therapy
Surgery
Primary Prevention
Secondary Prevention
Cost-Effectiveness of Therapy
Future or Investigational Therapies
Case Studies
Case #1
Congenital rubella syndrome pathophysiology On the Web
Most recent articles
Most cited articles
Review articles
CME Programs
Powerpoint slides
Images
American Roentgen Ray Society Images of Congenital rubella syndrome pathophysiology All Images X-rays Echo & Ultrasound CT Images MRI
Ongoing Trials at Clinical Trials.gov
US National Guidelines Clearinghouse
NICE Guidance
FDA on Congenital rubella syndrome pathophysiology
CDC on Congenital rubella syndrome pathophysiology
Congenital rubella syndrome pathophysiology in the news
Blogs on Congenital rubella syndrome pathophysiology
Directions to Hospitals Treating Congenital rubella syndrome
Risk calculators and risk factors for Congenital rubella syndrome pathophysiology

← Older edit Newer edit →

VisualWikitext

Revision as of 18:21, 16 January 2017

Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Dima Nimri, M.D. [2]

Overview

Pathophysiology

References

Template:WH Template:WS

@@ Line 1: / Line 1: @@
 __NOTOC__
-{{CMG}} {{AE}} {{KD}}
+{{CMG}}; {{AE}}{{DN}}
 {{Congenital rubella syndrome}}
 ==Overview==
-Congenital rubella syndrome results from the spread of infection to the [[fetus]] by the mother during the viremic stage. Rubella virus enters the fetus through the [[placenta]] and causes damage to all the germ layers of the fetus.  This results in rapid death of some cells.
 ==Pathophysiology==
-Congenital rubella syndrome results from the [[infection]] of fetus by [[transmission]] of virus from the mother during the viremic stage of the infection. Virus travels through the blood stream of the fetus and damages the blood vessels. This results in ischemic injury to the cells in the germ layers. The risk of congenital infection and defects is highest during the first 12 weeks of gestation and decreases after the 12th week of [[gestation]] with defects rare after the 20th week of gestation. If infection occurs 0–28 days before [[conception]], there is a 43% chance the infant will be affected. If the infection occurs 0–12 weeks after conception, there is a 51% chance the infant will be affected. If the infection occurs 13–26 weeks after conception there is a 23% chance the infant will be affected by the disease. Infants are not generally affected if [[rubella]] is contracted during the third trimester, or 26–40 weeks after conception. Problems rarely occur when rubella is contracted by the mother after 20 weeks of [[gestation]] and continues to disseminate the virus after birth.
 ==References==
 {{Reflist|2}}
-[[Category:Disease]]
-[[Category:Pediatrics]]
-[[Category:Neonatology]]
-[[Category:Congenital disorders]]
-[[Category:Infectious disease]]
-[[Category:Syndromes]]
 {{WH}}
 {{WS}}

Congenital rubella syndrome pathophysiology: Difference between revisions

Revision as of 18:21, 16 January 2017

Overview

Pathophysiology

References

Navigation menu

Search