Primary hyperaldosteronism causes: Difference between revisions
Jump to navigation
Jump to search
Usama Talib (talk | contribs) No edit summary |
|||
Line 1: | Line 1: | ||
__NOTOC__ | __NOTOC__ | ||
{{ | {{Primary hyperaldosteronism}} | ||
{{CMG}}; {{AE}}{{HK}} | {{CMG}}; {{AE}}{{HK}} | ||
==Overview== | ==Overview== |
Revision as of 15:07, 10 July 2017
Primary hyperaldosteronism Microchapters |
Differentiating Primary Hyperaldosteronism from other Diseases |
---|
Diagnosis |
Treatment |
Case Studies |
Primary hyperaldosteronism causes On the Web |
American Roentgen Ray Society Images of Primary hyperaldosteronism causes |
Risk calculators and risk factors for Primary hyperaldosteronism causes |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Syed Hassan A. Kazmi BSc, MD [2]
Overview
Causes
Common Causes
Common causes of Conn's Syndrome may be divided into:
- Adrenal causes:
- Aldosterone-secreting adrenal adenoma (APA-benign tumor, 50-60%)
- Idiopathic hyperaldosteronism (IHA-Bilateral hyperplasia of the adrenal gland, 40-50%)
- Extra-adrenal causes
- Ectopic secretion of aldosterone (Ovaries and Kidneys)
Less Common Causes
- Familial hyperaldosteronism type I (glucocorticoid-remediable aldosteronism [GRA])
- Familial hyperaldosteronism II (the familial occurrence of APA or bilateral idiopathic hyperplasia or both)
- Familial hyperaldosteronism type III (associated with the germline mutation in the KCNJ5 potassium channel)
- Pure aldosterone-producing adrenocortical carcinomas
- Unilateral adrenal hyperplasia