Hypoaldosteronism secondary prevention: Difference between revisions
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==Overview== | ==Overview== | ||
Effective measures for the secondary prevention of hypoaldosteronism include liberal salt intake of 4gm/day (to increase plasma sodium concentration), decreasing potassium intake and avoidance of drugs that affects renin angiotensin aldosterone system (RAAS) such as ACE inhibitors, ARBs, potassium sparing diuretics and β-Adrenergic receptor blockers. | Effective measures for the [[secondary prevention]] of hypoaldosteronism include liberal [[salt]] intake of 4gm/day (to increase [[plasma]] [[sodium]] [[concentration]]), decreasing [[potassium]] intake and avoidance of [[drugs]] that affects [[renin angiotensin aldosterone system]] ([[RAAS]]) such as [[ACE inhibitor|ACE inhibitors]], [[ARBs]], [[Potassium sparing diuretic|potassium sparing diuretics]] and [[Beta blockers|β-Adrenergic receptor blockers]]. | ||
==Secondary Prevention== | ==Secondary Prevention== | ||
*Effective measures for the secondary prevention of hypoaldosteronism include: | *Effective measures for the [[secondary prevention]] of hypoaldosteronism include: | ||
**Low potassium intake | **Low [[potassium]] intake | ||
**Salt intake of 4gm/day | **[[Salt]] intake of 4gm/day | ||
**Avoid drugs affecting the renin angiotensin aldosterone system (RAAS) such as: | **Avoid [[drugs]] affecting the [[renin angiotensin aldosterone system]] ([[RAAS]]) such as: | ||
***ACE inhibitors | ***[[ACE inhibitor|ACE inhibitors]] | ||
***Angiotensinogen receptor blocker | ***[[ARBs|Angiotensinogen receptor blocker (ARBs)]] | ||
***Potassium sparing diuretics | ***[[Potassium-sparing diuretic|Potassium sparing diuretics]] | ||
***β-Adrenergic receptor blockers | ***[[Beta blockers|β-Adrenergic receptor blockers]] | ||
==References== | ==References== |
Revision as of 14:42, 22 August 2017
Hypoaldosteronism Microchapters |
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Hypoaldosteronism secondary prevention On the Web |
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Akshun Kalia M.B.B.S.[2]
Overview
Effective measures for the secondary prevention of hypoaldosteronism include liberal salt intake of 4gm/day (to increase plasma sodium concentration), decreasing potassium intake and avoidance of drugs that affects renin angiotensin aldosterone system (RAAS) such as ACE inhibitors, ARBs, potassium sparing diuretics and β-Adrenergic receptor blockers.
Secondary Prevention
- Effective measures for the secondary prevention of hypoaldosteronism include:
- Low potassium intake
- Salt intake of 4gm/day
- Avoid drugs affecting the renin angiotensin aldosterone system (RAAS) such as: