Ogilvie syndrome laboratory findings: Difference between revisions
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{{Ogilvie syndrome}} | {{Ogilvie syndrome}} | ||
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==Overview== | ==Overview== | ||
Revision as of 20:12, 30 January 2018
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Ahmed Elsaiey, MBBCH [2]
Overview
An elevated/reduced concentration of serum/blood/urinary/CSF/other [lab test] is diagnostic of [disease name].
OR
Laboratory findings consistent with the diagnosis of [disease name] include [abnormal test 1], [abnormal test 2], and [abnormal test 3].
OR
[Test] is usually normal among patients with [disease name].
OR
Some patients with [disease name] may have elevated/reduced concentration of [test], which is usually suggestive of [progression/complication].
OR
There are no diagnostic laboratory findings associated with [disease name].
Laboratory Findings
- There are no specific diagnostic laboratory findings associated with Ogilvie's syndrome. The laboratory findings may include leukocytosis due to the underlying disease not due to the pseudo-obstruction itself.[1]
- In most of the patients metabolic imbalance may occur and it includes:[2][3]
- Hypokalemia
- Hypocalcemia
- Hypomagnesemia
- General laboratory tests are performed to exclude other causes and disease. The laboratory tests include the following:
- Complete blood count
- Electrolytes balance
- Lactate levels
- Thyroid hormones level
- To exclude acute abdominal pain diagnosis in patients suspected with perforation, the following laboratory tests are performed:
- Serum Aminotransferase
- Serum Alkaline phosphatase
- Bilirubin level
- Lipase level
- In patients with diarrhea, the following laboratory tests are performed:[4]
- Stool culture for Clostridia difficile
References
- ↑ Vanek VW, Al-Salti M (1986). "Acute pseudo-obstruction of the colon (Ogilvie's syndrome). An analysis of 400 cases". Dis Colon Rectum. 29 (3): 203–10. PMID 3753674.
- ↑ Jetmore AB, Timmcke AE, Gathright JB, Hicks TC, Ray JE, Baker JW (1992). "Ogilvie's syndrome: colonoscopic decompression and analysis of predisposing factors". Dis Colon Rectum. 35 (12): 1135–42. PMID 1473414.
- ↑ Sandle GI, Hunter M (2010). "Apical potassium (BK) channels and enhanced potassium secretion in human colon". QJM. 103 (2): 85–9. doi:10.1093/qjmed/hcp159. PMID 19892809.
- ↑ Simon M, Duong JP, Mallet V, Jian R, MacLennan KA, Sandle GI; et al. (2008). "Over-expression of colonic K+ channels associated with severe potassium secretory diarrhoea after haemorrhagic shock". Nephrol Dial Transplant. 23 (10): 3350–2. doi:10.1093/ndt/gfn411. PMID 18653901.