Behçet's disease pathophysiology: Difference between revisions

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Revision as of 01:32, 6 February 2018

Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Cafer Zorkun, M.D., Ph.D. [2]

Overview

Pathophysiology

Gross Pathology

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References

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 ==Overview==
-The underlying pathophysiological mechanism of Behçet's disease is an overactive [[immune system]] that produces recurrent outbreaks of inflammation in small blood vessels. Common symptoms include [[mouth ulcer]]s, [[genital ulcer]]s, [[eye]] inflammation, and [[arthritis]] in older patients.
 ==Pathophysiology==
-The symptoms of Behçet's disease are believed to be caused by an over-active [[immune system]] which, without any apparent [[infection]]s, produces recurrent outbreaks of inflammation in small blood vessels. Common symptoms include [[mouth ulcer]]s, [[genital ulcer]]s, [[eye]] inflammation, and [[arthritis]] in older patients. The symptoms are mostly painful but not life threatening. Some patients however experience significant impairment due to pain, impaired vision and reduced mobility, and may be disabled and unable to work due to this condition.  In some severe cases, uncontrolled inflammation may lead to [[blindness]], [[irritable bowel syndrome|intestinal complications]], [[stroke]], and even [[meningitis]], which can be fatal.
-Behçet's disease usually first affects patients when they are in their 20s and 30s. It then becomes a fluctuating lifelong disorder with a series of [[remission]]s and exacerbations which can last from days to months. Complete remission is rare.
 ==Gross Pathology==
-[[File:Behcet's syndrome 11.jpeg|Behcet's disease|500px]]
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 ==References==

Behçet's disease pathophysiology: Difference between revisions

Revision as of 01:32, 6 February 2018

Overview

Pathophysiology

Gross Pathology

References

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