Rapidly progressive glomerulonephritis risk factors: Difference between revisions
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* Pre existing renal disease | * Pre existing renal disease | ||
* Presence of HLA-DRB1, HLA DP1, DQ and DRB4s | * Presence of HLA-DRB1, HLA DP1, DQ and DRB4s<ref name="pmid15652778">{{cite journal| author=Jagiello P, Gross WL, Epplen JT| title=Complex genetics of Wegener granulomatosis. | journal=Autoimmun Rev | year= 2005 | volume= 4 | issue= 1 | pages= 42-7 | pmid=15652778 | doi=10.1016/j.autrev.2004.06.003 | pmc= | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=15652778 }} </ref> | ||
* Upper respiratory tract infection | * Upper respiratory tract infection | ||
* Sepsis | * Sepsis | ||
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Ahmed Elsaiey, MBBCH [2]
Overview
Common risk factors in the development of rapidly progressive glomerulonephritis may be occupational, environmental, genetic, and viral. ibodies t
Risk Factors
Common risk factors in the development of rapidly progressive glomerulonephritis may be occupational, environmental, genetic, and viral. [1][2]
Common risk factrors
- Pre existing renal disease
- Presence of HLA-DRB1, HLA DP1, DQ and DRB4s[3]
- Upper respiratory tract infection
- Sepsis
- Tobacco smoking
- IV drug abuse
Less common risk factors
- Cocaine use
- Exposure to hydrocarbons( formaldehyde)
- HIgh concenteration of FiO2
References
- ↑ Hellmark T, Segelmark M (2014). "Diagnosis and classification of Goodpasture's disease (anti-GBM)". J Autoimmun. 48-49: 108–12. doi:10.1016/j.jaut.2014.01.024. PMID 24456936.
- ↑ Bombassei GJ, Kaplan AA (1992). "The association between hydrocarbon exposure and anti-glomerular basement membrane antibody-mediated disease (Goodpasture's syndrome)". Am J Ind Med. 21 (2): 141–53. PMID 1536151.
- ↑ Jagiello P, Gross WL, Epplen JT (2005). "Complex genetics of Wegener granulomatosis". Autoimmun Rev. 4 (1): 42–7. doi:10.1016/j.autrev.2004.06.003. PMID 15652778.