Scrotal mass pathophysiology: Difference between revisions
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{{Scrotal mass}} | {{Scrotal mass}} | ||
{{CMG}}{{AE}} | {{CMG}}{{AE}}{{NE}} | ||
==Overview== | ==Overview== | ||
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===Physiology=== | ===Physiology=== | ||
===Phatogenesis=== | ===Phatogenesis=== | ||
==Genetics== | |||
==Associated Conditions== | |||
==Gros Pathology== | |||
==Microscopic Pathology== | |||
==Refrences== | ==Refrences== | ||
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[[Category:oncology,urology,surgery,radiology]] | |||
Revision as of 19:56, 30 October 2019
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Scrotal Mass Microchapters |
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Diagnosis |
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Treatment |
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Case Studies |
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Scrotal mass pathophysiology On the Web |
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American Roentgen Ray Society Images of Scrotal mass pathophysiology |
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Risk calculators and risk factors for Scrotal mass pathophysiology |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]Associate Editor(s)-in-Chief: Niloofarsadaat Eshaghhosseiny, MD[2]
Overview
Deponds on the causes phatophysiology is different.Inadequate fixation of the lower pole of the testis to the tunica vaginalis causes testicular tortion . If fixation is absent , the testis may torse (twist) on the spermatic cord, lead to produceischemia from reduced arterial inflow and venous outflow obstruction . Testicular torsion etiology include (eg, trauma, vigorous physical activity) or spontaneously. Acquired hernias due loss of mechanical integrity of the abdominal wall muscles and tendons . primary hernia due Genetic or systemic extracellular matrix disorders and defective wound healing after laparotomy and hernia repairs may predispose to incisional hernias.