Beriberi differential diagnosis: Difference between revisions
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!Laboratory findings | !Laboratory findings | ||
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!Cardiomyopathy due to other causes as alcohol or DM | |||
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* Idiopathic | * Idiopathic | ||
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* Elevated [[troponin]] (in ischemia) | * Elevated [[troponin]] (in ischemia) | ||
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!Delirium and delusional disorders | |||
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* [[Acute liver failure]] | * [[Acute liver failure]] | ||
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* Glucose and electrolyte levels | * Glucose and electrolyte levels | ||
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!Nerve entrapment disorders | |||
|Chronic injuries to nerves as they pass between bones and ligaments: | |Chronic injuries to nerves as they pass between bones and ligaments: | ||
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* MRI short inversion imaging recovery (STIR) technique | * MRI short inversion imaging recovery (STIR) technique | ||
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!Alcoholic hepatitis | |||
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* Chronic and excessive alcohol consumption | * Chronic and excessive alcohol consumption | ||
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* Liver US: Changes in liver size and dilatation of hepatic veins. | * Liver US: Changes in liver size and dilatation of hepatic veins. | ||
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!Diabetic ketoacidosis | |||
|In patients with type 1 DM, exposed to: | |In patients with type 1 DM, exposed to: | ||
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* Impaired renal function | * Impaired renal function | ||
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!Hyperthyroidism | |||
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* Primary: [[Graves' disease|Grave's disease]], toxic thyroid nodules and adenoma | * Primary: [[Graves' disease|Grave's disease]], toxic thyroid nodules and adenoma | ||
Revision as of 17:10, 7 November 2019
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Abdelrahman Ibrahim Abushouk, MD[2]
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Overview
Beriberi should be differentiated from other causes of cardiac dysfunction (wet beriberi), peripheral neuropathy (dry beriberi), delirium (WKS), and other disorders caused by excessive alcohol drinking. However, the differential diagnosis is broad due to the non-specific symptoms of cardiac and neural involvement associated with the condition.
Differentiating Beriberi from other Diseases
Beriberi should be differentiated from other causes of cardiac dysfunction (wet beriberi), peripheral neuropathy (dry beriberi), delirium (WKS), and other disorders caused by excessive alcohol drinking. However, the differential diagnosis is broad due to the non-specific symptoms of cardiac and neural involvement associated with beriberi.[1][2]
| Disorders | Etiology | Clinical Presentation | Laboratory findings |
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| Cardiomyopathy due to other causes as alcohol or DM |
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| Delirium and delusional disorders |
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| Nerve entrapment disorders | Chronic injuries to nerves as they pass between bones and ligaments:
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Within the distribution of the affected nerve, the patient may complain of:
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| Alcoholic hepatitis |
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| Diabetic ketoacidosis | In patients with type 1 DM, exposed to:
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| Hyperthyroidism |
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| Folic acid deficiency |
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References
- ↑ Whitfield KC, Bourassa MW, Adamolekun B, Bergeron G, Bettendorff L, Brown KH; et al. (2018). "Thiamine deficiency disorders: diagnosis, prevalence, and a roadmap for global control programs". Ann N Y Acad Sci. 1430 (1): 3–43. doi:10.1111/nyas.13919. PMC 6392124. PMID 30151974.
- ↑ DiNicolantonio JJ, Liu J, O'Keefe JH (2018). "Thiamine and Cardiovascular Disease: A Literature Review". Prog Cardiovasc Dis. 61 (1): 27–32. doi:10.1016/j.pcad.2018.01.009. PMID 29360523.